About: Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1

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About: Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies Goto Sponge NotDistinct Permalink

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description	article (en) ലേഖനം (ml) հոդված (hy) wetenschappelijk artikel (nl) article scientifique publié en 2004 (fr) наукова стаття, опублікована в серпні 2004 (uk) im Juli 2004 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q56876055-916AAE8C-50D1-4E6F-AB9F-2D982D543B51
publication date	2004-07-20 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q56876055-79EB487B-D698-48B7-BF96-B35420546D05
language of work or name	English
author name string	wds:Q56876055-149E0E4B-3B9B-445F-BCFB-11E391ABC217 wds:Q56876055-3910D4D7-28B7-425C-B84A-BA605E3B204C wds:Q56876055-5DA1F83B-C516-409D-849D-C83D128AE298 wds:Q56876055-707AAD75-A756-4833-B993-08BD3CFE8485 wds:Q56876055-92C5DC75-667B-45FE-BBB6-4935FD7CB038 wds:Q56876055-D51A7921-9B4B-4A6C-849D-D553EFF938CA wds:Q56876055-FC899888-1C69-4D63-AB32-E8B7D77390C8
author name string	Rodney E Phillips Andrew Johnson Charles Edwards Philippa J Easterbrook Anele Waters Hua-Tang Zhang Tin Tun
rdfs:label	Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (en) Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (nl)
skos:prefLabel	Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (en) Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (nl)
name	Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (en) Loss of Viral Control in Early HIV‐1 Infection Is Temporally Associated with Sequential Escape from CD8+T Cell Responses and Decrease in HIV–1–Specific CD4+and CD8+T Cell Frequencies (nl)
author	wds:Q56876055-2784DEE3-F1B6-4A4B-8C4B-366A4953B9FF wds:Q56876055-53CDC745-A8FD-412F-9DC2-024F24153E0B wds:Q56876055-7DBF3D78-4697-448A-A89B-E0F65E2D2E20 wds:Q56876055-7E870D79-D061-44AA-A0AB-3EB3F8E0F190 wds:Q56876055-B7CCF2DB-EC3E-476D-B346-B3E50CAB5EDB
author	Edward C. Holmes David A Price Annette Oxenius Emma Gostick Alexandra Trkola
title	wds:Q56876055-4984D259-4653-4B82-9928-F8D5DF87B73F
title	Loss of viral control in early HIV-1 infection is temporally associated with sequential escape from CD8+ T cell responses and decrease in HIV-1-specific CD4+ and CD8+ T cell frequencies (en)
page(s)	wds:Q56876055-2F8A8B47-FAF2-491F-9395-CED45F20D9AF
page(s)	713-721
instance of	wds:Q56876055-CD4EA72C-9C3C-452D-B838-7F37D3A1507E
instance of	scholarly article
PubMed ID	wds:Q56876055-9129411B-E187-43CA-83FA-EB45376A62BA
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15272399
PubMed ID	15272399
published in	wds:Q56876055-B4A8935A-2933-4E23-8A3B-09383D565BDA
published in	Journal of Infectious Diseases
issue	wds:Q56876055-7E056F6B-3B8B-4F07-B4C1-F09836D285D2
volume	wds:Q56876055-6433DE4D-A877-48A6-9250-427BDAC43D80
issue	4
volume	190
DOI	wds:Q56876055-D9B8A77C-7A2E-4AD1-88BB-9FEABD11DDA4
DOI	http://dx.doi.org/10.1086/422760
DOI	10.1086/422760
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q56876055
is cites work of	Prevention of Cytotoxic T Cell Escape Using a Heteroclitic Subdominant Viral T Cell Determinant Preemptive priming readily overcomes structure-based mechanisms of virus escape Apoptosis of HIV-specific CD8+ T cells: an HIV evasion strategy. Quantifying the impact of human immunodeficiency virus-1 escape from cytotoxic T-lymphocytes Sexual transmission of single human immunodeficiency virus type 1 virions encoding highly polymorphic multisite cytotoxic T-lymphocyte escape variants Human immunodeficiency virus type 1 clade B superinfection: evidence for differential immune containment of distinct clade B strains Correlates of protective cellular immunity revealed by analysis of population-level immune escape pathways in HIV-1. Intrapatient escape in the A0201-restricted epitope SLYNTVATL drives evolution of human immunodeficiency virus type 1 at the population level Evidence of differential HLA class I-mediated viral evolution in functional and accessory/regulatory genes of HIV-1 Human immunodeficiency virus type 1-specific CD8+ T-cell responses during primary infection are major determinants of the viral set point and loss of CD4+ T cells Rapid reversion of sequence polymorphisms dominates early human immunodeficiency virus type 1 evolution Retracing our STEP towards a successful CTL-based HIV-1 vaccine Role of Bim in regulating CD8+ T-cell responses during chronic viral infection. Structured observations reveal slow HIV-1 CTL escape Population genetic estimation of the loss of genetic diversity during horizontal transmission of HIV-1. Induction of multifunctional human immunodeficiency virus type 1 (HIV-1)-specific T cells capable of proliferation in healthy subjects by using a prime-boost regimen of DNA- and modified vaccinia virus Ankara-vectored vaccines expressing HIV-1 Gag c Low human immunodeficiency virus envelope diversity correlates with low in vitro replication capacity and predicts spontaneous control of plasma viremia after treatment interruptions Dynamic correlation between CTL response and viral load in primary human immunodeficiency virus-1 infected Koreans Virological outcome after structured interruption of antiretroviral therapy for human immunodeficiency virus infection is associated with the functional profile of virus-specific CD8+ T cells Structural and functional constraints limit options for cytotoxic T-lymphocyte escape in the immunodominant HLA-B27-restricted epitope in human immunodeficiency virus type 1 capsid Sequential broadening of CTL responses in early HIV-1 infection is associated with viral escape HIV-1 evolution following transmission to an HLA-B5801-positive patient Emergence of polyfunctional CD8+ T cells after prolonged suppression of human immunodeficiency virus replication by antiretroviral therapy Longitudinal assessment of human immunodeficiency virus type 1 (HIV-1)-specific gamma interferon responses during the first year of life in HIV-1-infected infants CTL quality and the control of human retroviral infections Primary human immunodeficiency virus type 1 (HIV-1) infection during HIV-1 Gag vaccination Maintenance or emergence of chronic phase secondary cytotoxic T lymphocyte responses after loss of acute phase immunodominant responses does not protect SIV-infected rhesus macaques from disease progression The evolutionary selective advantage of HIV-1 escape variants and the contribution of escape to the HLA-associated risk of AIDS progression Low CD8 T-cell proliferative potential and high viral load limit the effectiveness of therapeutic vaccination CTLA-4 blockade decreases TGF-beta, IDO, and viral RNA expression in tissues of SIVmac251-infected macaques Primary human immunodeficiency virus type 1 infection Clinical, virologic, and immunologic correlates of HIV-1 intraclade B dual infection among men who have sex with men. Impact of pre-adapted HIV transmission. Absence of SHIV infection in gut and lymph node tissues in rhesus monkeys after repeated rectal challenges following HIV-1 DNA/MVA immunizations Preservation HIV-1-specific IFNγ+ CD4+ T-cell responses in breakthrough infections after exposure to tenofovir gel in the CAPRISA 004 microbicide trial Immunization with adenovirus at the large intestinal mucosa as an effective vaccination strategy against sexually transmitted viral infection Variable processing and cross-presentation of HIV by dendritic cells and macrophages shapes CTL immunodominance and immune escape. Pol-Driven Replicative Capacity Impacts Disease Progression in HIV-1 Subtype C Infection Estimating the fitness cost of escape from HLA presentation in HIV-1 protease and reverse transcriptase.

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