About: José Antonio Pariente

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) forsker (nb) researcher (en) 연구원 (ko) 研究者 (ja) araştırmacı (tr) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq) ikertzaile espainiarra (eu) investigador espanhol (pt) investigador español (es) investigador español (gl) investigador español (ast) chercheur espagnol (fr) investigador espanyol (ca) ricercatore spagnolo (it) Spaans onderzoeker (nl)
rdfs:label	J.A. Pariente (es) J.A. Pariente (nl) J.A. Pariente (sl) J.A. Pariente (ast) José Antonio Pariente (ca) José Antonio Pariente (en) José Antonio Pariente (nb)
skos:prefLabel	J.A. Pariente (es) J.A. Pariente (nl) J.A. Pariente (sl) J.A. Pariente (ast) José Antonio Pariente (ca) José Antonio Pariente (en) José Antonio Pariente (nb)
name	J.A. Pariente (es) J.A. Pariente (nl) J.A. Pariente (sl) J.A. Pariente (ast) José Antonio Pariente (ca) José Antonio Pariente (en) José Antonio Pariente (nb)
Scopus author ID	wds:Q54054989-02218218-68AA-494B-BCC9-558155716B78
Scopus author ID	35586832400
given name	wds:Q54054989-4EC83E84-3D15-4ACB-9FAA-27BDC5B36F4C
given name	José Antonio
instance of	wds:Q54054989-46FD6B91-B7AC-4A4A-80AF-A24E3D221757
instance of	Homo sapiens
skos:altLabel	J.A. Pariente (en)
occupation	wds:Q54054989-18A840A7-BCF1-41AE-9309-53714073644A
employer	wds:Q54054989-A78746F9-AFFA-4B33-AC8F-BB239C69F6E8
languages spoken, written or signed	wds:Q54054989-F8024348-7CC8-414D-A3D5-10629061DC3F
sex or gender	wds:Q54054989-876B60AF-4CBE-41B8-89A0-3B5C520AE6B6
country of citizenship	wds:Q54054989-B65CA4B2-D352-42DF-AC6C-3B21FD90C6D3
occupation	researcher
employer	University of Extremadura
languages spoken, written or signed	Spanish
sex or gender	male
country of citizenship	Spain
ORCID iD	wds:Q54054989-7E659EA2-4F2C-4763-B022-8E099B8EF667
ORCID iD	https://orcid.org/0000-0002-9094-9943
ORCID iD	0000-0002-9094-9943
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q54054989
is author of	A role for 5,6-epoxyeicosatrienoic acid in calcium entry by de novo conformational coupling in human platelets. Free cytosolic calcium levels modify intracellular pH in rat pancreatic acini Ethanol impairs CCK-8-evoked amylase secretion through Ca2+-mediated ROS generation in mouse pancreatic acinar cells Melatonin counteracts alterations in oxidative metabolism and cell viability induced by intracellular calcium overload in human leucocytes: changes with age. H2O2 mobilizes Ca2+ from agonist- and thapsigargin-sensitive and insensitive intracellular stores and stimulates glutamate secretion in rat hippocampal astrocytes. Protective effect of melatonin against human leukocyte apoptosis induced by intracellular calcium overload: relation with its antioxidant actions Interaction of STIM1 with endogenously expressed human canonical TRP1 upon depletion of intracellular Ca2+ stores Melatonin as a potential tool against oxidative damage and apoptosis in ejaculated human spermatozoa. Exogenous melatonin supplementation prevents oxidative stress-evoked DNA damage in human spermatozoa. Melatonin protects human spermatozoa from apoptosis via melatonin receptor– and extracellular signal–regulated kinase-mediated pathways Melatonin reduces body weight gain and increases nocturnal activity in male Wistar rats. A role for SNAP-25 but not VAMPs in store-mediated Ca2+ entry in human platelets. Hydrogen peroxide increases the phagocytic function of human neutrophils by calcium mobilisation Effects of reactive oxygen species on actin filament polymerisation and amylase secretion in mouse pancreatic acinar cells. High endogenous melatonin concentrations enhance sperm quality and short-term in vitro exposure to melatonin improves aspects of sperm motility Thrombin induces activation and translocation of Bid, Bax and Bak to the mitochondria in human platelets. Autophagy-related proteins are functionally active in human spermatozoa and may be involved in the regulation of cell survival and motility. Thrombin induces apoptotic events through the generation of reactive oxygen species in human platelets. Early caspase-3 activation independent of apoptosis is required for cellular function Store-operated Ca2+ entry: vesicle fusion or reversible trafficking and de novo conformational coupling? Dual effect of hydrogen peroxide on store-mediated calcium entry in human platelets. Store-operated Ca(2+) entry and tyrosine kinase pp60(src) hyperactivity are modulated by hyperglycemia in platelets from patients with non insulin-dependent diabetes mellitus. Hydrogen peroxide and peroxynitrite enhance Ca2+ mobilization and aggregation in platelets from type 2 diabetic patients. Endogenously generated reactive oxygen species reduce PMCA activity in platelets from patients with non-insulin-dependent diabetes mellitus Differential involvement of thrombin receptors in Ca2+ release from two different intracellular stores in human platelets. SERCA2b activity is regulated by cyclophilins in human platelets. Role of proton gradients and vacuola H(+)-ATPases in the refilling of intracellular calcium stores in exocrine cells Differential involvement of vacuolar H(+)-ATPase in the refilling of thapsigargin- and agonist-mobilized Ca(2+) stores Intracellular Ca2+ homeostasis and aggregation in platelets are impaired by ethanol through the generation of H2O2 and oxidation of sulphydryl groups The inhibition of TNF-α-induced leucocyte apoptosis by melatonin involves membrane receptor MT1/MT2 interaction. Effect of exogenous melatonin on viability, ingestion capacity, and free-radical scavenging in heterophils from young and old ringdoves (Streptopelia risoria) Tryptophan Modulates Cell Viability, Phagocytosis and Oxidative Metabolism in Old Ringdoves Effect of xanthine oxidase-catalyzed reactive oxygen species generation on secretagogue-evoked calcium mobilization in mouse pancreatic acinar cells. Generation of ROS in response to CCK-8 stimulation in mouse pancreatic acinar cells. Changes in mitochondrial activity evoked by cholecystokinin in isolated mouse pancreatic acinar cells. H2O2-induced changes in mitochondrial activity in isolated mouse pancreatic acinar cells.

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