About: David H. Raulet

Facets (new session)
Description
Metadata
Settings
- owl:sameAs
- Inference Rule:

About: David H. Raulet Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

American immunologist

Attributes	Values
rdf:type	Item
description	American immunologist (en) immunoloog (nl)
rdfs:label	David H Raulet (es) David H Raulet (nl) David H. Raulet (en)
skos:prefLabel	David H Raulet (es) David H Raulet (nl) David H. Raulet (en)
name	David H Raulet (es) David H Raulet (nl) David H. Raulet (en)
National Academy of Sciences member ID	wds:Q51794345-d3d28eb7-4a4b-3f3f-9fa2-96d5f1ddaf94
National Academy of Sciences member ID	20047191
Scopus author ID	wds:Q51794345-85620422-E099-49E8-BD93-F28A95BBB7F2
Scopus author ID	7006111540
given name	wds:Q51794345-2DB81417-E310-47EE-BBBA-1D5D7D9300AA
given name	David
instance of	wds:Q51794345-4792B40F-84DB-4CF9-B58F-A8801946FC66
instance of	Homo sapiens
award received	wds:Q51794345-92a3bc84-4b71-1cd7-38d7-25366f31f35d
award received	William B. Coley Award
doctoral student	wds:Q51794345-f42b2c91-477b-942a-7d4e-26745cbc96d2
Google Knowledge Graph ID	wds:Q51794345-A1DB6933-4A8F-403F-8C41-A276CD85E573
Google Knowledge Graph ID	http://g.co/kg/g/11f0136fz7
doctoral student	Russell E. Vance
Google Knowledge Graph ID	/g/11f0136fz7
skos:altLabel	David Raulet (en)
occupation	wds:Q51794345-51ABF910-E8EC-42D3-AA53-6591094AE293
employer	wds:Q51794345-AA64E603-4AF2-4693-B8CD-6A28521B45A4
sex or gender	wds:Q51794345-BC5C8CED-8FAF-48EF-8E42-283246395D31
VIAF ID	wds:Q51794345-1d88394d-4038-c4de-7f69-e1d7864d99b2
Library of Congress authority ID	wds:Q51794345-6f5dafab-40b3-3a8e-cc97-171df5fb40e1
country of citizenship	wds:Q51794345-25cd6f7d-44fd-e8ad-9373-60c24ebaa9fb
educated at	wds:Q51794345-51791847-5B56-4813-9232-7302EF11C9D0
family name	wds:Q51794345-75153bf0-4713-e568-3986-50e746101832
VIAF ID	http://viaf.org/viaf/53589975
Library of Congress authority ID	http://id.loc.gov/authorities/names/n2006184514
occupation	immunologist
employer	University of California, Berkeley
sex or gender	male
VIAF ID	53589975
Library of Congress authority ID	n2006184514
country of citizenship	United States of America
educated at	Massachusetts Institute of Technology
family name	Raulet
ORCID iD	wds:Q51794345-DFC54AF5-30EE-48EA-B086-F4F5C6872AF4
ORCID iD	https://orcid.org/0000-0002-1257-8649
ORCID iD	0000-0002-1257-8649
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q51794345 David H. Raulet
is author of	The genomic arrangement of T cell receptor variable genes is a determinant of the developmental rearrangement pattern Gene placement and competition control T cell receptor gamma variable region gene rearrangement Development and selection of gammadelta T cells Redundant and unique roles of two enhancer elements in the TCRgamma locus in gene regulation and gammadelta T cell development Blastocyst MHC, a putative murine homologue of HLA-G, protects TAP-deficient tumor cells from natural killer cell-mediated rejection in vivo NKG2D mediates NK cell hyperresponsiveness and influenza-induced pathologies in a mouse model of chronic obstructive pulmonary disease Functionally conformed free class I heavy chains exist on the surface of beta 2 microglobulin negative cells NK cells respond to pulmonary infection with Mycobacterium tuberculosis, but play a minimal role in protection. The role of the NKG2D immunoreceptor in immune cell activation and natural killing Roles of the NKG2D immunoreceptor and its ligands Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease. Genomic Ly49A transgenes: basis of variegated Ly49A gene expression and identification of a critical regulatory element NK cells developing in vitro from fetal mouse progenitors express at least one member of the Ly49 family that is acquired in a time-dependent and stochastic manner independently of CD94 and NKG2. The developmental fate of T cells is critically influenced by TCRgammadelta expression. A differentiation factor required for the expression of cytotoxic T-cell function Missing self-recognition of Ocil/Clr-b by inhibitory NKR-P1 natural killer cell receptors Diversity, rearrangement, and expression of murine T cell gamma genes Selective associations with signaling proteins determine stimulatory versus costimulatory activity of NKG2D Comparative analysis of human NK cell activation induced by NKG2D and natural cytotoxicity receptors Innate or adaptive immunity? The example of natural killer cells Cutting edge: tumor rejection mediated by NKG2D receptor-ligand interaction is dependent upon perforin Antitumor immunity. A shed NKG2D ligand that promotes natural killer cell activation and tumor rejection NK1.1+ T cells in the liver arise in the thymus and are selected by interactions with class I molecules on CD4+CD8+ cells. The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor Self-tolerance of natural killer cells Positive selection of dendritic epidermal gammadelta T cell precursors in the fetal thymus determines expression of skin-homing receptors Oncogenic stress sensed by the immune system: role of natural killer cell receptors NK cell self tolerance, responsiveness and missing self recognition Rae1 and H60 ligands of the NKG2D receptor stimulate tumour immunity A sense of something missing Expression of the RAE-1 family of stimulatory NK-cell ligands requires activation of the PI3K pathway during viral infection and transformation The MHC reactivity of the T cell repertoire prior to positive and negative selection. Multiplicity and plasticity of natural killer cell signaling pathways Developmental regulation of T-cell receptor gene expression A novel ligand for the NKG2D receptor activates NK cells and macrophages and induces tumor immunity The DNA damage response arouses the immune system Regulation of ligands for the NKG2D activating receptor RAE-1 ligands for the NKG2D receptor are regulated by E2F transcription factors, which control cell cycle entry Expression and function of NK cell receptors in CD8+ T cells Clonal acquisition of inhibitory Ly49 receptors on developing NK cells is successively restricted and regulated by stromal class I MHC. Innate immune recognition by stimulatory immunoreceptors Binding of diverse peptides to MHC class I molecules inhibits target cell lysis by activated natural killer cells Major histocompatibility complex class I-dependent skewing of the natural killer cell Ly49 receptor repertoire. Major histocompatibility complex genes determine natural killer cell tolerance. DNA mismanagement leads to immune system oversight MHC class I-deficient mice Inhibition of MHC class I is a virulence factor in herpes simplex virus infection of mice Events that regulate differentiation of alpha beta TCR+ and gamma delta TCR+ T cells from a common precursor. Stromal-cell regulation of natural killer cell differentiation

Faceted Search & Find service v1.16.117 as of May 05 2024

Alternative Linked Data Documents: ODE Content Formats:

RDF

ODATA

Microdata

About

OpenLink Virtuoso version 07.20.3239 as of May 5 2024, on Linux (x86_64-centos_6-linux-gnu), Single-Server Edition (378 GB total memory, 193 GB memory in use)
Data on this page belongs to its respective rights holders.
Virtuoso Faceted Browser Copyright © 2009-2024 OpenLink Software