About: Nicholas A. Wright

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher (ORCID 0000-0002-5525-3457)

Attributes	Values
rdf:type	Item
description	연구원 (ko) 研究者 (ja) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq) researcher (ORCID 0000-0002-5525-3457) (en)
rdfs:label	Nicholas A. Wright (en) Nicholas A Wright (nl) Nicholas A Wright (sl) Nicholas A. Wright (cs) Nicholas A. Wright (es)
skos:prefLabel	Nicholas A. Wright (en) Nicholas A Wright (nl) Nicholas A Wright (sl) Nicholas A. Wright (cs) Nicholas A. Wright (es)
name	Nicholas A. Wright (en) Nicholas A Wright (nl) Nicholas A Wright (sl) Nicholas A. Wright (cs) Nicholas A. Wright (es)
Scopus author ID	wds:Q50690182-D866C74E-A073-46F8-822E-9541E5145ADD
Scopus author ID	7201531909
given name	wds:Q50690182-BCCEF7E8-F3AA-4072-BAD1-FD3EFF6A156E
given name	Nicholas
instance of	wds:Q50690182-5733D31A-601A-45E0-B9AC-E66A8BEDE32A
NL CR AUT ID	wds:Q50690182-09F3DB84-7DAC-4867-B633-5AB05553279E
instance of	Homo sapiens
NL CR AUT ID	xx0074472
skos:altLabel	Nicholas Wright (en)
occupation	wds:Q50690182-72FE1200-7994-4B65-B04B-D1CC182834E5
employer	wds:Q50690182-1EDE2B2C-0E60-4048-9347-9DAB1450DAB3
sex or gender	wds:Q50690182-8FF6BBD4-BA9A-4B02-A1FC-C9985F42439A
VIAF ID	wds:Q50690182-24A20441-0C78-4857-A25A-1ABA750BCEED
GND ID	wds:Q50690182-f6a303d9-e69c-4f4c-9425-e18c74ad69b7
date of birth	wds:Q50690182-63E63D17-1401-46DC-AE99-B966E9CD5891
educated at	wds:Q50690182-00B0D8E9-B096-4377-ADB8-86E8B5A21A36
family name	wds:Q50690182-1B210B6E-5F7E-4FD4-8832-3BF928E0B063
official website	wds:Q50690182-4267F1E6-C1DE-48AC-8D51-07D25DD18CD9
VIAF ID	http://viaf.org/viaf/100289892
GND ID	https://d-nb.info/gnd/1058653474
occupation	researcher
employer	Barts and The London School of Medicine and Dentistry
sex or gender	male
VIAF ID	100289892
GND ID	1058653474
date of birth	1943-01-01 00:00:00Z (xsd:dateTime)
educated at	Newcastle University
family name	Wright
official website	http://www.bci.qmul.ac.uk/staff/item/wright
ORCID iD	wds:Q50690182-09C6C0D9-F043-4831-802F-A9D308AD9C66
ORCID iD	https://orcid.org/0000-0002-5525-3457
ORCID iD	0000-0002-5525-3457
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q50690182
is author of	Analysis of the clonal architecture of the human small intestinal epithelium establishes a common stem cell for all lineages and reveals a mechanism for the fixation and spread of mutations. Gastrointestinal stem cells and cancer: bridging the molecular gap. Expansion of a mutated clone: from stem cell to tumour. The gastrointestinal tract stem cell niche Bone marrow contribution to tumor-associated myofibroblasts and fibroblasts. The sources of parenchymal regeneration after chronic hepatocellular liver injury in mice. Proteinase activated receptor 1 mediated fibrosis in a mouse model of liver injury: a role for bone marrow derived macrophages Locating the stem cell niche and tracing hepatocyte lineages in human liver The histogenesis of regenerative nodules in human liver cirrhosis Recipes for adult stem cell plasticity: fusion cuisine or readymade? Expression of annexin VI (p68, 67 kDa-calelectrin) in normal human tissues: evidence for developmental regulation in B- and T-lymphocytes. X-inactivation patch size in human female tissue confounds the assessment of tumor clonality A significant proportion of myofibroblasts are of bone marrow origin in human liver fibrosis. Bone marrow contributes to renal parenchymal turnover and regeneration Stem cell relationships and the origin of gastrointestinal cancer. Functional role of CD44v-xCT system in the development of spasmolytic polypeptide-expressing metaplasia. Circulating mesenchymal stem cells. Epithelial stem cell repertoire in the gut: clues to the origin of cell lineages, proliferative units and cancer Mechanisms of field cancerization in the human stomach: the expansion and spread of mutated gastric stem cells Spasmolytic polypeptide-expressing metaplasia and intestinal metaplasia: time for reevaluation of metaplasias and the origins of gastric cancer. The stem cells of small intestinal crypts: where are they? Haematopoietic lineage-committed bone marrow cells, but not cloned cultured mesenchymal stem cells, contribute to regeneration of renal tubular epithelium after HgCl 2 -induced acute tubular injury The cellular origin and proliferative status of regenerating renal parenchyma after mercuric chloride damage and erythropoietin treatment. Hepatocytes from non-hepatic adult stem cells Bone marrow–derived liver stem cells: Their therapeutic potential A regenerative role for bone marrow following experimental colitis: contribution to neovasculogenesis and myofibroblasts. A methodological approach to tracing cell lineage in human epithelial tissues Lrig1 controls intestinal stem-cell homeostasis by negative regulation of ErbB signalling Breast cancer dormancy can be maintained by small numbers of micrometastases. Coordinated localisation of mucins and trefoil peptides in the ulcer associated cell lineage and the gastrointestinal mucosa. Aspects of the biology of regeneration and repair in the human gastrointestinal tract. Subcellular distribution of peptides associated with gastric mucosal healing and neoplasia. Glicentin, an active enteroglucagon, has a significant trophic role on the small intestine but not on the colon in the rat Colonic subepithelial myofibroblasts in mucosal inflammation and repair: contribution of bone marrow-derived stem cells to the gut regenerative response. Age-associated mitochondrial DNA mutations lead to small but significant changes in cell proliferation and apoptosis in human colonic crypts Mitochondrial DNA mutations are established in human colonic stem cells, and mutated clones expand by crypt fission. The gastrointestinal stem cell Top down or bottom up? Competing management structures in the morphogenesis of colorectal neoplasms. Lineage tracing reveals multipotent stem cells maintain human adenomas and the pattern of clonal expansion in tumor evolution. Field cancerization in the GI tract. Stem cells and cancer: an intimate relationship.

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