About: Larissa Fabritz

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) গবেষক (bn) 研究者 (ja) araştırmacı (tr) Kardiologin (de) wetenschapper (nl) дослідниця (uk) 研究者 (zh) հետազոտող (hy) hulumtuese (sq)
rdfs:label	Larissa Fabritz (de) Larissa Fabritz (en) Larissa Fabritz (es) Larissa Fabritz (fr) Larissa Fabritz (nl) Larissa Fabritz (sl) Larissa Fabritz (ast) Larissa Fabritz (bar)
skos:prefLabel	Larissa Fabritz (de) Larissa Fabritz (en) Larissa Fabritz (es) Larissa Fabritz (fr) Larissa Fabritz (nl) Larissa Fabritz (sl) Larissa Fabritz (ast) Larissa Fabritz (bar)
name	Larissa Fabritz (de) Larissa Fabritz (en) Larissa Fabritz (es) Larissa Fabritz (fr) Larissa Fabritz (nl) Larissa Fabritz (sl) Larissa Fabritz (ast) Larissa Fabritz (bar)
Scopus author ID	wds:Q47714033-C799EDCC-7F17-4EB4-9A8A-50E7728ADBFA
Scopus author ID	6602628929
given name	wds:Q47714033-40C54CC5-9FD9-4B87-858C-EA2CCBB15D5A
given name	Larissa
instance of	wds:Q47714033-73BCD55A-FBF3-4EB5-AF87-2B2225DBC372
instance of	Homo sapiens
Loop ID	wds:Q47714033-181B06C8-5B51-4AA7-97FF-D3A0E3535B2D
Loop ID	26592
Hamburger Professo...essorenkatalog ID	wds:Q47714033-5C4AA2FB-B803-4E6F-B25F-9DFE61E77E51
Hamburger Professo...essorenkatalog ID	00005894
occupation	wds:Q47714033-6B854C4C-9004-452D-B3D3-3A841BD2AFB4
employer	wds:Q47714033-B61A762E-1AC7-4081-AE5C-253615D9B1FB
sex or gender	wds:Q47714033-A66459D6-A90E-40FD-842F-049D341AC08E
ISNI	wds:Q47714033-EE34B2F6-1970-430D-AEB9-608F245FCBBC
VIAF ID	wds:Q47714033-E850E2EF-EA64-4DF7-AB96-3FB539873445
GND ID	wds:Q47714033-B40A4DF2-067C-4022-8A55-038FF844EF5E
date of birth	wds:Q47714033-3A3398B6-936D-40BC-A34C-B1BF35D9874E
VIAF ID	http://viaf.org/viaf/25439856
GND ID	https://d-nb.info/gnd/120665018
occupation	researcher
employer	University of Birmingham
sex or gender	female organism
ISNI	0000000015967493
VIAF ID	25439856
GND ID	120665018
date of birth	2000-01-01 00:00:00Z (xsd:dateTime)
ORCID iD	wds:Q47714033-4587B131-0A2C-425F-8A9E-8D5E54ED15B8
ORCID iD	https://orcid.org/0000-0002-9241-1733
ORCID iD	0000-0002-9241-1733
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q47714033
is author of	Familial hypertrophic cardiomyopathy-linked mutant troponin T causes stress-induced ventricular tachycardia and Ca2+-dependent action potential remodeling. Survivin determines cardiac function by controlling total cardiomyocyte number Autonomic modulation and antiarrhythmic therapy in a model of long QT syndrome type 3 Vascular endothelium is critically involved in the hypotensive and hypovolemic actions of atrial natriuretic peptide Arrhythmogenic left atrial cellular electrophysiology in a murine genetic long QT syndrome model PITX2c Is Expressed in the Adult Left Atrium, and Reducing Pitx2c Expression Promotes Atrial Fibrillation Inducibility and Complex Changes in Gene Expression Proarrhythmia in a non-failing murine model of cardiac-specific Na+/Ca 2+ exchanger overexpression: whole heart and cellular mechanisms Shock-induced dispersion of ventricular repolarization: implications for the induction of ventricular fibrillation and the upper limit of vulnerability Ventricular arrhythmias, increased cardiac calmodulin kinase II expression, and altered repolarization kinetics in ANP receptor deficient mice Age- and Training-Dependent Development of Arrhythmogenic Right Ventricular Cardiomyopathy in Heterozygous Plakoglobin-Deficient Mice Ca/calmodulin kinase II differentially modulates potassium currents Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels Popeye domain containing proteins are essential for stress-mediated modulation of cardiac pacemaking in mice Overexpression of cAMP-response element modulator causes abnormal growth and development of the atrial myocardium resulting in a substrate for sustained atrial fibrillation in mice Systematic analysis of gene expression differences between left and right atria in different mouse strains and in human atrial tissue Universal cardiac induction of human pluripotent stem cells in two and three-dimensional formats: implications for in vitro maturation. Minimally invasive closed-chest ultrasound-guided substance delivery into the pericardial space in mice Constitutively active phosphatase inhibitor-1 improves cardiac contractility in young mice but is deleterious after catecholaminergic stress and with aging Divergent proarrhythmic potential of macrolide antibiotics despite similar QT prolongation: fast phase 3 repolarization prevents early afterdepolarizations and torsade de pointes Local Atrial Natriuretic Peptide Signaling Prevents Hypertensive Cardiac Hypertrophy in Endothelial Nitric-oxide Synthase-deficient Mice Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality Flecainide acetate for the treatment of atrial and ventricular arrhythmias Cardiac pacemaker function of HCN4 channels in mice is confined to embryonic development and requires cyclic AMP. Hypervolemic hypertension in mice with systemic inactivation of the (floxed) guanylyl cyclase-A gene by alphaMHC-Cre-mediated recombination Enhanced activity of the myocardial Na+/H+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice. Heart-directed expression of a human cardiac isoform of cAMP-response element modulator in transgenic mice Late Sodium Current in Human Atrial Cardiomyocytes from Patients in Sinus Rhythm and Atrial Fibrillation Phytanic acid accumulation is associated with conduction delay and sudden cardiac death in sterol carrier protein-2/sterol carrier protein-x deficient mice Insights into sick sinus syndrome from an inducible mouse model. The European Network for Translational Research in Atrial Fibrillation (EUTRAF): objectives and initial results An introduction to murine models of atrial fibrillation A Regional Reduction in Ito and IKACh in the Murine Posterior Left Atrial Myocardium Is Associated with Action Potential Prolongation and Increased Ectopic Activity

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