About: Frederick M. Hecht

Facets (new session)
Description
Metadata
Settings
- owl:sameAs
- Inference Rule:

About: Frederick M. Hecht Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

biomedical researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) 연구원 (ko) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) изилдөөчү (ky) 研究者 (zh) biomedical researcher (en)
rdfs:label	Frederick M. Hecht (en) Frederick M. Hecht (es) Frederick M. Hecht (nl) Frederick M. Hecht (sl) Frederick M. Hecht (ast) فريدريك إم. هيكت (ar)
skos:prefLabel	Frederick M. Hecht (en) Frederick M. Hecht (es) Frederick M. Hecht (nl) Frederick M. Hecht (sl) Frederick M. Hecht (ast) فريدريك إم. هيكت (ar)
name	Frederick M. Hecht (en) Frederick M. Hecht (es) Frederick M. Hecht (nl) Frederick M. Hecht (sl) Frederick M. Hecht (ast) فريدريك إم. هيكت (ar)
Scopus author ID	wds:Q45955550-09E6EE2E-F7EA-47EA-8406-3D2D2083AF92
Scopus author ID	7103064306
given name	wds:Q45955550-3FEB40D1-3006-4D63-9E44-EF9E83278BEF
given name	Frederick
instance of	wds:Q45955550-112E9FBF-48A2-4238-B8E7-D2D1E48E1AF7
instance of	Homo sapiens
Google Scholar author ID	wds:Q45955550-355d8d9e-4b42-03b6-d976-e1a74063365e
Google Scholar author ID	BapD9LwAAAAJ
skos:altLabel	Frederick Hecht (en) Rick Hecht (en)
occupation	wds:Q45955550-572443FB-999E-4359-9F40-D532CAB16448
employer	wds:Q45955550-5085517d-4c47-64ba-4e10-6f5ebb49d751
sex or gender	wds:Q45955550-FEE149CB-A1C1-460F-B8D6-2E9519DC6CCC
VIAF ID	wds:Q45955550-7E5FC847-0AA2-40DE-AEB9-865078BA621C
Library of Congress authority ID	wds:Q45955550-C3D4B6FE-7F72-46DF-98D9-87D2D0BE0A16
educated at	wds:Q45955550-cecc242a-4386-291f-bcb9-585b1d08c22c
family name	wds:Q45955550-17D09CDF-B6D7-4145-9AA5-758F2CC04A6B
WorldCat Identities ID (superseded)	wds:Q45955550-4F314601-D201-4AAF-8CBE-D36ABDE485EF
VIAF ID	http://viaf.org/viaf/21269681
Library of Congress authority ID	http://id.loc.gov/authorities/names/n89138631
occupation	researcher
employer	University of California, San Francisco
sex or gender	male
VIAF ID	21269681
Library of Congress authority ID	n89138631
educated at	SUNY Downstate Health Sciences University
family name	Hecht
WorldCat Identities ID (superseded)	lccn-n89138631
ORCID iD	wds:Q45955550-CD011BCE-E1D8-4655-86E9-5B1C162F6E43
Dimensions author ID	wds:Q45955550-4AE67636-EC1D-4B70-829B-CE4B645D9512
ORCID iD	https://orcid.org/0000-0002-5782-1171
ORCID iD	0000-0002-5782-1171
Dimensions author ID	012511713057.20
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q45955550
is author of	Synergy or independence? Deciphering the interaction of HLA Class I and NK cell KIR alleles in early HIV-1 disease progression Human CD4+ CD25+ regulatory T cells control T-cell responses to human immunodeficiency virus and cytomegalovirus antigens Adherence to protease inhibitors, HIV-1 viral load, and development of drug resistance in an indigent population. Body awareness: construct and self-report measures Mindfulness-based stress reduction for HIV treatment side effects: a randomized, wait-list controlled trial Symptom management with massage and acupuncture in postoperative cancer patients: a randomized controlled trial. Cellular immune responses and viral diversity in individuals treated during acute and early HIV-1 infection. Independent assessment of candidate HIV incidence assays on specimens in the CEPHIA repository Persistence of primary drug resistance among recently HIV-1 infected adults A multicenter observational study of the potential benefits of initiating combination antiretroviral therapy during acute HIV infection Characterization of human immunodeficiency virus type 1 populations containing CXCR4-using variants from recently infected individuals New DNA viruses identified in patients with acute viral infection syndrome. New testing strategy to detect early HIV-1 infection for use in incidence estimates and for clinical and prevention purposes. CCL3L1 and CCR5 influence cell-mediated immunity and affect HIV-AIDS pathogenesis via viral entry-independent mechanisms Low proportions of CD28- CD8+ T cells expressing CD57 can be reversed by early ART initiation and predict mortality in treated HIV infection Emergence of individual HIV-specific CD8 T cell responses during primary HIV-1 infection can determine long-term disease outcome Impact of HIV on CD8+ T cell CD57 expression is distinct from that of CMV and aging Rapid progressing allele HLA-B35 Px restricted anti-HIV-1 CD8+ T cells recognize vestigial CTL epitopes Conferral of enhanced natural killer cell function by KIR3DS1 in early human immunodeficiency virus type 1 infection The effects of early antiretroviral therapy and its discontinuation on the HIV-specific antibody response. The relation between symptoms, viral load, and viral load set point in primary HIV infection. Antiretroviral therapy initiated within 6 months of HIV infection is associated with lower T-cell activation and smaller HIV reservoir size The HIV-1 reservoir in eight patients on long-term suppressive antiretroviral therapy is stable with few genetic changes over time Seroreversion in subjects receiving antiretroviral therapy during acute/early HIV infection Influence of HLA-B57 on clinical presentation and viral control during acute HIV-1 infection. Reduction in CD8+ cell noncytotoxic anti-HIV activity in individuals receiving highly active antiretroviral therapy during primary infection. Neutralizing antibody responses against autologous and heterologous viruses in acute versus chronic human immunodeficiency virus (HIV) infection: evidence for a constraint on the ability of HIV to completely evade neutralizing antibody responses Human immunodeficiency virus type 1-specific CD8+ T-cell responses during primary infection are major determinants of the viral set point and loss of CD4+ T cells Limited HIV infection of central memory and stem cell memory CD4+ T cells is associated with lack of progression in viremic individuals Incomplete peripheral CD4+ cell count restoration in HIV-infected patients receiving long-term antiretroviral treatment Seroconversion following nonoccupational postexposure prophylaxis against HIV. Central nervous system immune activation characterizes primary human immunodeficiency virus 1 infection even in participants with minimal cerebrospinal fluid viral burden Select host restriction factors are associated with HIV persistence during antiretroviral therapy Cytomegalovirus-specific T cells persist at very high levels during long-term antiretroviral treatment of HIV disease Relative dominance of epitope-specific cytotoxic T-lymphocyte responses in human immunodeficiency virus type 1-infected persons with shared HLA alleles Expansion of CD7(low) and CD7(negative) CD8 T-cell effector subsets in HIV-1 infection: correlation with antigenic load and reversion by antiretroviral treatment. Microbial translocation is a cause of systemic immune activation in chronic HIV infection

Faceted Search & Find service v1.16.117 as of May 05 2024

Alternative Linked Data Documents: ODE Content Formats:

RDF

ODATA

Microdata

About

OpenLink Virtuoso version 07.20.3239 as of May 5 2024, on Linux (x86_64-centos_6-linux-gnu), Single-Server Edition (378 GB total memory, 188 GB memory in use)
Data on this page belongs to its respective rights holders.
Virtuoso Faceted Browser Copyright © 2009-2024 OpenLink Software