About: Marco Crescenzi

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) ricercatore (it) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) araştırmacı (tr) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq) onderzoeker (nl)
rdfs:label	Marco Crescenzi (en) Marco Crescenzi (es) Marco Crescenzi (nl) Marco Crescenzi (sl) Marco Crescenzi (ast)
skos:prefLabel	Marco Crescenzi (en) Marco Crescenzi (es) Marco Crescenzi (nl) Marco Crescenzi (sl) Marco Crescenzi (ast)
name	Marco Crescenzi (en) Marco Crescenzi (es) Marco Crescenzi (nl) Marco Crescenzi (sl) Marco Crescenzi (ast)
Scopus author ID	wds:Q43280564-76A8D998-5373-4710-B7EE-DBEAA03E139E
Scopus author ID	7004350369
given name	wds:Q43280564-A07A6934-642C-45B0-ADC4-0660D14499F8
given name	Marco
instance of	wds:Q43280564-59C4E38B-1A7C-42DB-90A9-A66156802320
instance of	Homo sapiens
occupation	wds:Q43280564-B96C0E13-3C28-4A14-AB41-7A30BF5FAC35
Parsifal cluster ID	wds:Q43280564-B0BF8555-939D-4E8B-8850-E8EACD73D56B
sex or gender	wds:Q43280564-584CDD0B-1A3B-4635-9228-EA147AB21E7E
ISNI	wds:Q43280564-3E5467A9-15F6-43C6-BAF7-F30F4439A894
VIAF ID	wds:Q43280564-59625AC9-BCB8-4994-B224-A60C576637BB
Library of Congress authority ID	wds:Q43280564-504CFBAB-D1C1-48DD-BD20-034DF3A9ACA1
IdRef ID	wds:Q43280564-FEFB44C8-75DF-4695-A870-4AFEF6801E64
WorldCat Identities ID (superseded)	wds:Q43280564-291F0EFB-3EA8-4036-9024-405CBBCB2B26
VIAF ID	http://viaf.org/viaf/6747333
Library of Congress authority ID	http://id.loc.gov/authorities/names/n2001129502
IdRef ID	http://www.idref.fr/07067471X/id
occupation	researcher
Parsifal cluster ID	88013
sex or gender	male
ISNI	0000000047936413
VIAF ID	6747333
Library of Congress authority ID	n2001129502
IdRef ID	07067471X
WorldCat Identities ID (superseded)	lccn-n2001129502
ORCID iD	wds:Q43280564-D2F43C04-52F5-40E3-ACF8-52125ACCAC46
ORCID iD	https://orcid.org/0000-0003-0156-1494
ORCID iD	0000-0003-0156-1494
ResearcherID	wds:Q43280564-EDCFF6AB-C11F-49AA-A66E-F8E1FCB9F480
ResearcherID	J-3603-2018
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q43280564
is author of	Che-1 phosphorylation by ATM/ATR and Chk2 kinases activates p53 transcription and the G2/M checkpoint Wild-type p53 induces diverse effects in 32D cells expressing different oncogenes. Terminally differentiated muscle cells are defective in base excision DNA repair and hypersensitive to oxygen injury. Critical requirement for cell cycle inhibitors in sustaining nonproliferative states. Robust G2 pausing of adult stem cells in Hydra The main biological determinants of tumor line taxonomy elucidated by a principal component analysis of microarray data. New functions of XPC in the protection of human skin cells from oxidative damage The nebulin SH3 domain is dispensable for normal skeletal muscle structure but is required for effective active load bearing in mouse. Increased levels of acute-phase inflammatory proteins in plasma of patients with sporadic CJD. The mammalian mismatch repair pathway removes DNA 8-oxodGMP incorporated from the oxidized dNTP pool The mammalian mismatch repair protein MSH2 is required for correct MRE11 and RAD51 relocalization and for efficient cell cycle arrest induced by ionizing radiation in G2 phase Inhibition of ErbB-2 mitogenic and transforming activity by RALT, a mitogen-induced signal transducer which binds to the ErbB-2 kinase domain Alpha 6 beta 4 and alpha 6 beta 1 integrins associate with ErbB-2 in human carcinoma cell lines. A pRb-independent mechanism preserves the postmitotic state in terminally differentiated skeletal muscle cells. The accumulation of MMS-induced single strand breaks in G1 phase is recombinogenic in DNA polymerase beta defective mammalian cells Phosphorylation on threonine 11 of β-dystrobrevin alters its interaction with kinesin heavy chain p53 re-expression inhibits proliferation and restores differentiation of human thyroid anaplastic carcinoma cells. HIPK2 sustains apoptotic response by phosphorylating Che-1/AATF and promoting its degradation Differentiation-associated microRNAs antagonize the Rb-E2F pathway to restrict proliferation. A role for oxidized DNA precursors in Huntington's disease-like striatal neurodegeneration Terminally differentiated skeletal myotubes are not confined to G0 but can enter G1 upon growth factor stimulation Gene expression waves. Cell cycle independent collective dynamics in cultured cells. Expression of E1A in terminally differentiated muscle cells reactivates the cell cycle and suppresses tissue-specific genes by separable mechanisms Knockdown of cyclin-dependent kinase inhibitors induces cardiomyocyte re-entry in the cell cycle Mismatch repair, G(2)/M cell cycle arrest and lethality after DNA damage. Megalencephalic leukoencephalopathy with subcortical cysts protein-1 modulates endosomal pH and protein trafficking in astrocytes: relevance to MLC disease pathogenesis Sensitivity to DNA cross-linking chemotherapeutic agents in mismatch repair-defective cells in vitro and in xenografts Anti-GAPDH Autoantibodies as a Pathogenic Determinant and Potential Biomarker of Neuropsychiatric Diseases Hypogammaglobulinemia with hyper-IgM, severe T-cell defect, and abnormal recirculation of OKT4 lymphocytes in a girl with chronic lymphadenopathy. Np95 is regulated by E1A during mitotic reactivation of terminally differentiated cells and is essential for S phase entry Phosphorylation and nitration of tyrosine residues affect functional properties of Synaptophysin and Dynamin I, two proteins involved in exo-endocytosis of synaptic vesicles Phosphorylation of SRSF1 is modulated by replicational stress Peroxynitrite induces tyrosine residue modifications in synaptophysin C-terminal domain, affecting its interaction with src Cytogenetic analysis of human cells reveals specific patterns of DNA damage in replicative and oncogene-induced senescence Streptococcal-vimentin cross-reactive antibodies induce microvascular cardiac endothelial proinflammatory phenotype in rheumatic heart disease. Wt-p53 action in human leukaemia cell lines corresponding to different stages of differentiation. Trends in tissue repair and regeneration

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