About: Pamela Kreeger

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

bioengineering researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) 연구원 (ko) গবেষক (bn) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідниця (uk) эрдэмтэн (mn) باحت (ary) 研究者 (zh) հետազոտող (hy) hulumtuese (sq) bioengineering researcher (en)
rdfs:label	Pamela Kreeger (en) Pamela Kreeger (es) Pamela Kreeger (nl) Pamela Kreeger (sl) Pamela Kreeger (ast)
skos:prefLabel	Pamela Kreeger (en) Pamela Kreeger (es) Pamela Kreeger (nl) Pamela Kreeger (sl) Pamela Kreeger (ast)
name	Pamela Kreeger (en) Pamela Kreeger (es) Pamela Kreeger (nl) Pamela Kreeger (sl) Pamela Kreeger (ast)
given name	wds:Q43187851-4127365D-2FDA-493F-B0EF-77063D9B8471
given name	Pamela
instance of	wds:Q43187851-273E25BB-F8C1-48D1-8C42-678E9B9214C7
instance of	Homo sapiens
award received	wds:Q43187851-916bf2a0-44cc-0790-3ba5-f275036151b8 wds:Q43187851-94eaf273-4aba-2b55-7da7-7b510b5b3010
award received	Fellow of the American Institute for Medical and Biological Engineering Fellow of the Biomedical Engineering Society
Academic Tree ID	wds:Q43187851-5C91D91E-075C-4081-B936-32C569793EF5
Academic Tree ID	255835
Google Scholar author ID	wds:Q43187851-a3445679-45e5-4969-1b2b-cd4b5bbb7d6e
Google Scholar author ID	C4SBJooAAAAJ
skos:altLabel	Pamela K Kreeger (en) Pamela K. Kreeger (en)
occupation	wds:Q43187851-D1C8BB0A-139E-4BFD-B590-A912EC486E45
employer	wds:Q43187851-729F91EB-02A3-4056-A7E3-0AA88E56FB72 wds:Q43187851-9646B2F6-9235-49BE-8B3F-322E91AFB8D9
sex or gender	wds:Q43187851-5954C573-A1A9-44E0-82BF-D8B21669C15C
on focus list of Wikimedia project	wds:Q43187851-3F8E29F8-5CE3-4358-8C6B-7C79A1C6FE2A
educated at	wds:Q43187851-981F0985-8666-4DB1-8D42-1D73A255918D wds:Q43187851-BC7DF074-DC88-475E-B04F-5E4221FF7980
family name	wds:Q43187851-5B2D4FE7-605B-4E95-BF10-40592A0E1547
occupation	researcher
employer	University of Wisconsin–Madison Massachusetts Institute of Technology
sex or gender	female organism
on focus list of Wikimedia project	WikiProject PCC Wikidata Pilot/Northwestern University Libraries
educated at	Northwestern University Valparaiso University
family name	Kreeger
ORCID iD	wds:Q43187851-5EE2FA20-959E-4C23-BE38-A54EEF048D6E
ORCID iD	https://orcid.org/0000-0001-8193-1007
ORCID iD	0000-0001-8193-1007
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q43187851
is author of	Tissue-engineered follicles produce live, fertile offspring Cancer systems biology: a network modeling perspective Postnatal regulation of germ cells by activin: the establishment of the initial follicle pool. Fate of the initial follicle pool: empirical and mathematical evidence supporting its sufficiency for adult fertility. The in vitro regulation of ovarian follicle development using alginate-extracellular matrix gels Regulation of mouse follicle development by follicle-stimulating hormone in a three-dimensional in vitro culture system is dependent on follicle stage and dose Murine granulosa cell morphology and function are regulated by a synthetic Arg-Gly-Asp matrix RAS mutations affect tumor necrosis factor-induced apoptosis in colon carcinoma cells via ERK-modulatory negative and positive feedback circuits along with non-ERK pathway effects Strategies from UW-Madison for rescuing biomedical research in the US. Experimental and computational analysis of cellular interactions with nylon-3-bearing substrates Analysis of the quantitative balance between insulin-like growth factor (IGF)-1 ligand, receptor, and binding protein levels to predict cell sensitivity and therapeutic efficacy Integration of multiple signaling pathway activities resolves K-RAS/N-RAS mutation paradox in colon epithelial cell response to inflammatory cytokine stimulation Using partial least squares regression to analyze cellular response data Alternatively-Activated Macrophages Upregulate Mesothelial Expression of P-Selectin to Enhance Adhesion of Ovarian Cancer Cells Decoupling the effects of stiffness and fiber density on cellular behaviors via an interpenetrating network of gelatin-methacrylate and collagen. Ovarian Cells Have Increased Proliferation in Response to Heparin-Binding Epidermal Growth Factor as Collagen Density Increases Analysis of cancer signaling networks by systems biology to develop therapies. Design and implementation of a microfluidic device capable of temporal growth factor delivery reveal filtering capabilities of the EGFR/ERK pathway Hierarchy of cellular decisions in collective behavior: Implications for wound healing A multivariate model of ErbB network composition predicts ovarian cancer cell response to canertinib Partial Least Squares Regression Models for the Analysis of Kinase Signaling M2 macrophages induce ovarian cancer cell proliferation via a heparin binding epidermal growth factor/matrix metalloproteinase 9 intercellular feedback loop Reaction of Morpholine with t-Butyl Acetoacetate: A Study in Kinetic vs Thermodynamic Control, Product Identification, and Molecular Modeling Quantitative analysis of insulin-like growth factor 2 receptor and insulin-like growth factor binding proteins to identify control mechanisms for insulin-like growth factor 1 receptor phosphorylation A kinetic model identifies phosphorylated estrogen receptor-α (ERα) as a critical regulator of ERα dynamics in breast cancer Alternatively activated macrophage-derived secretome stimulates ovarian cancer spheroid spreading through a JAK2/STAT3 pathway Immobilized epidermal growth factor stimulates persistent, directed keratinocyte migration via activation of PLCγ1 A simplified culture system to examine soluble factor interactions between mammalian cells High-grade serous ovarian cancer cell lines exhibit heterogeneous responses to growth factor stimulation Engineering Approaches to Study Cellular Decision-Making. Endometriotic Epithelial Cell Response to Macrophage-Secreted Factors is Dependent on Extracellular Matrix Context Engineering the Extracellular Matrix to Model the Evolving Tumor Microenvironment Scaffold stiffness influences breast cancer cell invasion via EGFR-linked Mena upregulation and matrix remodeling Substrate curvature induces fallopian tube epithelial cell invasion via cell-cell tension in a model of ovarian cortical inclusion cysts The Extracellular Matrix of Ovarian Cortical Inclusion Cysts Modulates Invasion of Fallopian Tube Epithelial Cells Ten simple rules for women principal investigators during a pandemic

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