About: Karim Fouad

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	ikertzailea (eu) researcher (en) 연구원 (ko) গবেষক (bn) 研究者 (ja) araştırmacı (tr) дослідниця (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) onderzoeker (nl) hulumtuese (sq)
rdfs:label	Karim Fouad (en) Karim Fouad (es) Karim Fouad (nl) Karim Fouad (sl) Karim Fouad (ast)
skos:prefLabel	Karim Fouad (en) Karim Fouad (es) Karim Fouad (nl) Karim Fouad (sl) Karim Fouad (ast)
name	Karim Fouad (en) Karim Fouad (es) Karim Fouad (nl) Karim Fouad (sl) Karim Fouad (ast)
Scopus author ID	wds:Q41685186-F14045A2-395A-4B14-BBEB-719EFF0E17AD
Scopus author ID	7003972001
given name	wds:Q41685186-877208A5-CE2F-4A18-9560-0EC8844AE2C6
given name	Karim
instance of	wds:Q41685186-3F1CD986-88C6-4CF9-81B4-F0BBBC59270C
instance of	Homo sapiens
occupation	wds:Q41685186-19EB513A-7F4B-4432-B697-1BE5DB2160F8
employer	wds:Q41685186-F4C66BFB-908B-4231-AEDA-97288E3698FF
sex or gender	wds:Q41685186-4D139640-FEE7-4470-A4A9-41A2404F663A
VIAF ID	wds:Q41685186-8a1b99ec-546c-4c37-9b75-77d4a3797953
GND ID	wds:Q41685186-28a6680f-a02a-4126-85e9-cd3e71cd31de
educated at	wds:Q41685186-078F027C-41D3-423F-B058-F5806EC2AA34
family name	wds:Q41685186-280A6A7F-DD4E-43A2-9123-0A4A851F66BD
VIAF ID	http://viaf.org/viaf/8991155769104527880001
GND ID	https://d-nb.info/gnd/1185653333
occupation	researcher
employer	University of Alberta
sex or gender	female organism
VIAF ID	8991155769104527880001
GND ID	1185653333
educated at	University of Konstanz
family name	Fouad
ORCID iD	wds:Q41685186-5BB98B50-2481-4567-BCF2-8AB526ECF381
ORCID iD	https://orcid.org/0000-0003-3654-7852
ORCID iD	0000-0003-3654-7852
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q41685186
is author of	Anatomical correlates of locomotor recovery following dorsal and ventral lesions of the rat spinal cord. Training of Walking Skills Overground and on the Treadmill: Case Series on Individuals With Incomplete Spinal Cord Injury Anatomical correlates of recovery in single pellet reaching in spinal cord injured rats Comment on "Restoring voluntary control of locomotion after paralyzing spinal cord injury". Electrical stimulation of intact peripheral sensory axons in rats promotes outgrowth of their central projections. Functional switch between motor tracts in the presence of the mAb IN-1 in the adult rat. Nogo-A antibody improves regeneration and locomotion of spinal cord-injured rats Regenerating corticospinal fibers in the Marmoset (Callitrix jacchus) after spinal cord lesion and treatment with the anti-Nogo-A antibody IN-1. Treadmill training in incomplete spinal cord injured rats. Efficient testing of motor function in spinal cord injured rats. Red nucleus projections to distinct motor neuron pools in the rat spinal cord. Locomotion after spinal cord injury depends on constitutive activity in serotonin receptors A new electrode configuration for recording electromyographic activity in behaving mice. Neuronal coordination of arm and leg movements during human locomotion Restoring walking after spinal cord injury White matter glucose metabolism during intracortical electrostimulation: a quantitative [(18)F]Fluorodeoxyglucose autoradiography study in the rat. Modification of group I field potentials in the intermediate nucleus of the cat spinal cord after chronic axotomy of an extensor nerve Neuronal populations capable of regeneration following a combined treatment in rats with spinal cord transection Cervical sprouting of corticospinal fibers after thoracic spinal cord injury accompanies shifts in evoked motor responses Reaching training in rats with spinal cord injury promotes plasticity and task specific recovery. Synergistic effects of BDNF and rehabilitative training on recovery after cervical spinal cord injury. BDNF promotes connections of corticospinal neurons onto spared descending interneurons in spinal cord injured rats. Spontaneous locomotor recovery in spinal cord injured rats is accompanied by anatomical plasticity of reticulospinal fibers. Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors BDNF: the career of a multifaceted neurotrophin in spinal cord injury Combining Schwann cell bridges and olfactory-ensheathing glia grafts with chondroitinase promotes locomotor recovery after complete transection of the spinal cord Plasticity beyond peri-infarct cortex: spinal up regulation of structural plasticity, neurotrophins, and inflammatory cytokines during recovery from cortical stroke. Reorganization of descending motor tracts in the rat spinal cord Behavioral and electromyographic characterization of mice lacking EphA4 receptors. A systematic review of directly applied biologic therapies for acute spinal cord injury Motor axonal regeneration after partial and complete spinal cord transection Enhancement and resetting of locomotor activity by muscle afferents. Functional testing in animal models of spinal cord injury: not as straight forward as one would think Restoration of sensorimotor functions after spinal cord injury Spinal cord injury and plasticity: opportunities and challenges Improving axonal growth and functional recovery after experimental spinal cord injury by neutralizing myelin associated inhibitors Reticulospinal plasticity after cervical spinal cord injury in the rat involves withdrawal of projections below the injury. Reflex adaptations during treadmill walking with increased body load. Electromyographic activity associated with spontaneous functional recovery after spinal cord injury in rats. Plasticity after spinal cord injury: relevance to recovery and approaches to facilitate it Object recognition memory in zebrafish. Advantages of delaying the onset of rehabilitative reaching training in rats with incomplete spinal cord injury Training-induced plasticity in rats with cervical spinal cord injury: effects and side effects The unilateral 6-OHDA rat model of Parkinson's disease revisited: an electromyographic and behavioural analysis Ganglioside GM1 induces phosphorylation of mutant huntingtin and restores normal motor behavior in Huntington disease mice

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