About: Mizuko Mamura

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) ricercatore (it) গবেষক (bn) 研究者 (ja) araştırmacı (tr) chercheur (fr) дослідник (uk) эрдэмтэн (mn) 研究者 (zh) հետազոտող (hy) onderzoeker (nl) researcher (en-ca) researcher (en-gb) researcher (en-us) investigador (ca) investigador (gl) investigador (ast) investigador (pt) investigador/a (es)
rdfs:label	Mizuko Mamura (en) Mizuko Mamura (es) Mizuko Mamura (nl) Mizuko Mamura (sl) Mizuko Mamura (ast) 真村瑞子 (ja)
skos:prefLabel	Mizuko Mamura (en) Mizuko Mamura (es) Mizuko Mamura (nl) Mizuko Mamura (sl) Mizuko Mamura (ast) 真村瑞子 (ja)
name	Mizuko Mamura (en) Mizuko Mamura (es) Mizuko Mamura (nl) Mizuko Mamura (sl) Mizuko Mamura (ast) 真村瑞子 (ja)
Scopus author ID	wds:Q39976895-57AAEEE5-1BC3-4AC3-876E-5F46930F435D
Scopus author ID	6602433291
instance of	wds:Q39976895-D20904D0-9692-462A-8D9E-DB4F6A056564
instance of	Homo sapiens
Loop ID	wds:Q39976895-A526B887-D6BB-4A75-852F-F79EE2FE5345
Loop ID	240459
occupation	wds:Q39976895-FEEF2259-8F98-4B45-AA93-310C3AB4E982
employer	wds:Q39976895-6F9C753A-45D5-45CB-9545-B1ADED98E6CA wds:Q39976895-93709520-936D-4169-B47D-19974982EB05
CiNii Research ID	wds:Q39976895-C549B56A-F020-4AC3-B5A2-186EED3858C2
educated at	wds:Q39976895-AEF5A651-94AF-4C11-A81C-A0A73BCE89DC wds:Q39976895-EB097259-1E8D-435B-8E2D-3D46E3D4E9EF
CiNii Research ID	https://cir.nii.ac.jp/crid/1420282801188376832.rdf
occupation	researcher
employer	Kyungpook National University School of Medicine Tokyo Medical University
CiNii Research ID	1420282801188376832
educated at	Chiba University
ORCID iD	wds:Q39976895-08CC54D4-B443-47A2-8C62-E2BBBD3FA5A9
ORCID iD	https://orcid.org/0000-0003-4531-0144
ORCID iD	0000-0003-4531-0144
e-Rad researcher number	wds:Q39976895-C6C47673-20C4-4FD5-A928-5EAD415FA869
e-Rad researcher number	60400686
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q39976895
is author of	Loss of Smad3 in acute T-cell lymphoblastic leukemia. Activin receptor-like kinase5 inhibition suppresses mouse melanoma by ubiquitin degradation of Smad4, thereby derepressing eomesodermin in cytotoxic T lymphocytes Lack of a role for transforming growth factor-beta in cytotoxic T lymphocyte antigen-4-mediated inhibition of T cell activation. Raloxifene, a mixed estrogen agonist/antagonist, induces apoptosis through cleavage of BAD in TSU-PR1 human cancer cells Lifetime exposure to a soluble TGF-beta antagonist protects mice against metastasis without adverse side effects Transforming growth factor-beta production and myeloid cells are an effector mechanism through which CD1d-restricted T cells block cytotoxic T lymphocyte-mediated tumor immunosurveillance: abrogation prevents tumor recurrence. Smad4 signalling in T cells is required for suppression of gastrointestinal cancer. An anti-transforming growth factor beta antibody suppresses metastasis via cooperative effects on multiple cell compartments. Overexpression of phosphorylated STAT-1alpha in the labial salivary glands of patients with Sjögren's syndrome. Interleukin-17 gene expression in patients with rheumatoid arthritis. Role of increased penile expression of transforming growth factor-beta1 and activation of the Smad signaling pathway in erectile dysfunction in streptozotocin-induced diabetic rats Crucial role of the interleukin-6/interleukin-17 cytokine axis in the induction of arthritis by glucose-6-phosphate isomerase Transforming growth factor-beta can suppress tumorigenesis through effects on the putative cancer stem or early progenitor cell and committed progeny in a breast cancer xenograft model Tumor necrosis factor induces apoptosis in hepatoma cells by increasing Ca(2+) release from the endoplasmic reticulum and suppressing Bcl-2 expression IN-1130, a novel transforming growth factor-beta type I receptor kinase (activin receptor-like kinase 5) inhibitor, promotes regression of fibrotic plaque and corrects penile curvature in a rat model of Peyronie's disease Anti-interleukin-6 receptor antibody (tocilizumab) treatment of multicentric Castleman's disease Inhibition of erythropoiesis by Smad6 in human cord blood hematopoietic stem cells. Tumor necrosis factor-alpha mediates antiapoptotic signals partially via p38 MAP kinase activation in human eosinophils CD28 disruption exacerbates inflammation in Tgf-beta1-/- mice: in vivo suppression by CD4+CD25+ regulatory T cells independent of autocrine TGF-beta1. Association of variants in PPARγ², IGF2BP2, and KCNQ1 with a susceptibility to gestational diabetes mellitus in a Korean population. TGF-beta1 production by CD4+ CD25+ regulatory T cells is not essential for suppression of intestinal inflammation. Proangiogenic TIE2(+)/CD31 (+) macrophages are the predominant population of tumor-associated macrophages infiltrating metastatic lymph nodes Blockade of TGF-beta signaling in T cells prevents the development of experimental glomerulonephritis Inhibition of lethal inflammatory responses through the targeting of membrane-associated Toll-like receptor 4 signaling complexes with a Smad6-derived peptide. Significance of antiprothrombin antibodies in patients with systemic lupus erythematosus: clinical evaluation of the antiprothrombin assay and the antiphosphatidylserine/prothrombin assay, and comparison with other antiphospholipid antibody assays Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation Absence of Smad3 induces neutrophil migration after cutaneous irradiation: possible contribution to subsequent radioprotection Constitutive phosphorylation and nuclear localization of Smad3 are correlated with increased collagen gene transcription in activated hepatic stellate cells Pathophysiological roles of cytokine-chemokine immune network Identification of three novel peptides that inhibit CD40-CD154 interaction Successful treatment of refractory thrombotic thrombocytopenic purpura with cyclosporine and corticosteroids in a patient with systemic lupus erythematosus and antibodies to ADAMTS13. Erratum to: Proangiogenic TIE2+/CD31+ Macrophages Are the Predominant Population of Tumor-Associated Macrophages Infiltrating Metastatic Lymph Nodes. Intravascular large B-cell lymphoma with acute abdomen as a presenting symptom in a patient with systemic lupus erythematosus XXIV World Allergy Congress 2015 Ligation of the T cell receptor complex results in phosphorylation of Smad2 in T lymphocytes.

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