About: Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test

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About: Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test Goto Sponge NotDistinct Permalink

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description	article scientifique publié en 2006 (fr) artículu científicu espublizáu en 2006 (ast) vědecký článek publikovaný v roce 2006 (cs) 2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած (hyw) наукова стаття, опублікована в серпні 2006 (uk) مقالة علمية (نشرت في أغسطس 2006) (ar) 2006 թվականի օգոստոսին հրատարակված գիտական հոդված (hy) vedecký článok (publikovaný 2006/08/01) (sk) wetenschappelijk artikel (gepubliceerd op 2006/08/01) (nl) scientific journal article (en)
publication date	wds:Q28595067-6F0CB05A-2246-4C39-9212-20A93AC511CC
publication date	2006-08-01 00:00:00Z (xsd:dateTime)
cites work	wds:Q28595067-7C705E83-9B17-4916-9EDE-80D29BED8C13
cites work	5,10-Methylenetetrahydrofolate reductase gene variants and congenital anomalies: a HuGE review
author name string	wds:Q28595067-6A8EB9B2-6EA4-491F-A0F5-354A67823607 wds:Q28595067-8DB49CCE-9ED6-47D0-AA41-95E7026CC17F
author name string	Diana M. Juriloff Muriel J. Harris
rdfs:label	Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (en) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (nl) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (ast)
skos:prefLabel	Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (en) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (nl) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (ast)
name	Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (en) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (nl) Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (ast)
author	wds:Q28595067-A71F3B3C-5ACD-40B7-BC90-7E16AEEAE5A4 wds:Q28595067-E7F71DB9-99FE-4CDD-BF96-12F4DAB02F8E wds:Q28595067-ED4F4135-1DFE-4E77-9455-C1435C2B3F8E
author	Thomas J. Carroll Andrew Paul McMahon Andrew C Lidral
title	wds:Q28595067-34E71B8B-0ED0-4A8B-9FA0-A989CC70B36F
title	Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation test (en)
page(s)	wds:Q28595067-24E4F2F4-31F7-4DC3-8EBD-17C36C1CD0E5
page(s)	574–579
instance of	wds:Q28595067-ED9811C7-E091-4166-B838-E79248E244F3
instance of	scholarly article
main subject	wds:Q28595067-75180925-4E73-4A8C-AC63-C8E14AC887A2 wds:Q28595067-D2A2C1A5-A8EA-42B6-883D-5A57AEC1481A wds:Q28595067-F0842499-3238-4C2B-B805-C53BC0E79753
main subject	Wingless-type MMTV integration site family, member 9B cleft palate cleft lip with or without cleft palate
PubMed ID	wds:Q28595067-C29E6EB4-D42F-4603-933F-3221755D41FB
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/16998816
PubMed ID	16998816
published in	wds:Q28595067-39B29166-F826-4922-AE6B-EAEDA3560BC4
published in	Birth Defects Research Part A: Clinical and Molecular Teratology
issue	wds:Q28595067-21C958E1-08B5-41B3-ACB1-1FC91CE22355
volume	wds:Q28595067-6D6A85CF-55A8-41C6-9FFB-3B21C2536764
issue	8
volume	76
DOI	wds:Q28595067-AB3644FA-EE61-418F-8219-299EEDE913D6
DOI	http://dx.doi.org/10.1002/BDRA.20302
DOI	10.1002/BDRA.20302
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28595067
is cites work of	Temporal-spatial ablation of neural crest in the mouse results in cardiovascular defects The etiopathogenesis of cleft lip and cleft palate: usefulness and caveats of mouse models Variation in WNT genes is associated with non-syndromic cleft lip with or without cleft palate Phenotypic variability and craniofacial dysmorphology: increased shape variance in a mouse model for cleft lip. Cleft lip and palate: understanding genetic and environmental influences Palatogenesis: engineering, pathways and pathologies Wnt/beta-catenin signaling in oral tissue development and disease Deciphering the Palimpsest: Studying the Relationship Between Morphological Integration and Phenotypic Covariation Pax6 regulates craniofacial form through its control of an essential cephalic ectodermal patterning center. Epigenetic integration of the developing brain and face Mouse resources for craniofacial research Role of GSK-3β in the osteogenic differentiation of palatal mesenchyme Cranial neural crest cells on the move: their roles in craniofacial development. Planar cell polarity signaling in craniofacial development Tissue engineering in cleft palate and other congenital malformations. A conserved Pbx-Wnt-p63-Irf6 regulatory module controls face morphogenesis by promoting epithelial apoptosis Expression of WNT signalling pathway genes during chicken craniofacial development. Frizzled 1 and frizzled 2 genes function in palate, ventricular septum and neural tube closure: general implications for tissue fusion processes Regulatory variant in FZD6 gene contributes to nonsyndromic cleft lip and palate in an African-American family Studies with Wnt genes and nonsyndromic cleft lip and palate. Expression of Gpr177, a Wnt trafficking regulator, in mouse embryogenesis The canonical Wnt signaling activator, R-spondin2, regulates craniofacial patterning and morphogenesis within the branchial arch through ectodermal-mesenchymal interaction Hear the Wnt Ror: how melanoma cells adjust to changes in Wnt. Genetics of nonsyndromic orofacial clefts A Comprehensive Overview of Skeletal Phenotypes Associated with Alterations in Wnt/β-catenin Signaling in Humans and Mice Examination of a palatogenic gene program in zebrafish Influence of maternal metabolism and parental genetics on fetal maldevelopment in diabetic rat pregnancy New directions in craniofacial morphogenesis Palate morphogenesis: current understanding and future directions Expression of Wnts in the developing murine secondary palate Wnt9b-dependent FGF signaling is crucial for outgrowth of the nasal and maxillary processes during upper jaw and lip development Determinants of orofacial clefting I: Effects of 5-Aza-2'-deoxycytidine on cellular processes and gene expression during development of the first branchial arch. Expression and mutation analyses implicate ARHGAP29 as the etiologic gene for the cleft lip with or without cleft palate locus identified by genome-wide association on chromosome 1p22. Expression of Wnt9, TCTP, and Bmp1/Tll in sea cucumber visceral regeneration. Distinct requirements for wnt9a and irf6 in extension and integration mechanisms during zebrafish palate morphogenesis Requirement for frzb and fzd7a in cranial neural crest convergence and extension mechanisms during zebrafish palate and jaw morphogenesis. Association of WNT9B Gene Polymorphisms With Nonsyndromic Cleft Lip With or Without Cleft Palate in Brazilian Nuclear Families. Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain Wnt/β-catenin signaling and Msx1 promote outgrowth of the maxillary prominences Tfap2a-dependent changes in mouse facial morphology result in clefting that can be ameliorated by a reduction in Fgf8 gene dosage. Further evidence suggesting a role for variation in ARHGAP29 variants in nonsyndromic cleft lip/palate.

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