About: Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1

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rdf:type	Item
description	artículu científicu espublizáu en 1992 (ast) vědecký článek publikovaný v roce 1992 (cs) 1992 թվականի մայիսին հրատարակված գիտական հոդված (hy) مقالة علمية (نشرت في مايو 1992) (ar) 1992 թուականի Մայիսին հրատարակուած գիտական յօդուած (hyw) наукова стаття, опублікована в травні 1992 (uk) vedecký článok (publikovaný 1992/05/01) (sk) scientific journal article (en) wetenschappelijk artikel (gepubliceerd op 1992/05/01) (nl) im Mai 1992 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q28591590-2EA568CB-48CA-4C1E-A278-C5A26AE81A40
publication date	1992-05-01 00:00:00Z (xsd:dateTime)
author name string	wds:Q28591590-3A7FF9E1-2548-45E6-BFA3-38E95364DECA wds:Q28591590-69F50216-8CE5-44D7-BD86-6F9184CB726C wds:Q28591590-AD2EBFF2-DC36-4F75-8F61-DA5DD665AA8E
author name string	D. W. Nebert A. Puga F. Carrier
rdfs:label	Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (en) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (nl) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (ast)
skos:prefLabel	Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (en) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (nl) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (ast)
name	Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (en) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (nl) Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (ast)
title	wds:Q28591590-8AE02F8A-63DD-4668-9296-7A87E0CE1CFB
title	Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1 (en)
page(s)	wds:Q28591590-A677768B-BD1C-43CA-9060-4699BB437F8D
page(s)	269–281
instance of	wds:Q28591590-228E811C-472F-427F-8E01-A2638DBEC0B8
instance of	scholarly article
main subject	wds:Q28591590-3829F007-874E-46DA-879D-5BB6F48F5CBE
main subject	Aryl-hydrocarbon receptor
PubMed ID	wds:Q28591590-07B114CD-56BF-4345-9B2F-B31ABB53580A
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/1605850
PubMed ID	1605850
published in	wds:Q28591590-BD866B26-0CFE-4A0F-944F-5DB83273B189
published in	DNA and Cell Biology
issue	wds:Q28591590-9E813539-B481-4E35-B352-0350C6E7B28C
volume	wds:Q28591590-5F8FD8D1-1DF7-414D-A90B-A314BC2EDE0D
issue	4
volume	11
DOI	wds:Q28591590-6C90459C-C1D2-48B8-9D1F-D548C3D671BC
DOI	http://dx.doi.org/10.1089/DNA.1992.11.269
DOI	10.1089/DNA.1992.11.269
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28591590
is cites work of	Modulation of oestrogen receptor signalling by association with the activated dioxin receptor The long winding road toward understanding the molecular mechanisms for B-cell suppression by 2,3,7,8-tetrachlorodibenzo-p-dioxin Protein kinase C modulates regulation of the CYP1A1 gene by the aryl hydrocarbon receptor Aryl hydrocarbon receptor-mediated signal transduction Activator protein 1 (AP-1)- and nuclear factor kappaB (NF-kappaB)-dependent transcriptional events in carcinogenesis Role of the aromatic hydrocarbon receptor and [Ah] gene battery in the oxidative stress response, cell cycle control, and apoptosis Epigenetic mechanisms of chemical carcinogenesis Lesions of aryl-hydrocarbon receptor-deficient mice Dioxins, the aryl hydrocarbon receptor and the central regulation of energy balance Ah Receptor Pathway Intricacies; Signaling Through Diverse Protein Partners and DNA-Motifs TCDD induces c-jun expression via a novel Ah (dioxin) receptor-mediated p38-MAPK-dependent pathway. The transcriptional signature of dioxin in human hepatoma HepG2 cells Epigenetic theories of cancer initiation 2,3,7,8-Tetrachlorodibenzo-p-dioxin treatment induces c-Fos expression in the forebrain of the Long-Evans rat. Sustained increase in intracellular free calcium and activation of cyclooxygenase-2 expression in mouse hepatoma cells treated with dioxin Phenobarbital induction of CYP1A1 gene expression in a primary culture of rainbow trout hepatocytes. Evidence for a second pathway in the action mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Significance of Ah-receptor mediated activation of protein kinase under cell-free conditions Constitutive activation of the aromatic hydrocarbon receptor Possible involvement of aryl hydrocarbon receptor (AhR) in adult T-cell leukemia (ATL) leukemogenesis: constitutive activation of AhR in ATL Antioxidant response element-mediated 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induction of human NAD(P)H:quinone oxidoreductase 1 gene expression Glutathione S-transferases: gene structure and regulation of expression Induction of human prostaglandin endoperoxide H synthase-2 (PHS-2) mRNA by TCDD. Expression of the aryl hydrocarbon receptor/transcription factor (AhR) and AhR-regulated CYP1 gene transcripts in a rat model of mammary tumorigenesis The Ah receptor is not involved in 2,3,7,8-tetrachlorodibenzo- p-dioxin-mediated apoptosis in human leukemic T cell lines Role of the aryl hydrocarbon receptor in cell cycle regulation Functional characterization and role of INrf2 in antioxidant response element-mediated expression and antioxidant induction of NAD(P)H:quinone oxidoreductase1 gene Induction of cytochrome P4501 as an indicator of potential chemical carcinogenesis On the significance of the role of cellular stress response reactions in the toxic actions of dioxin Regulation of constitutive and inducible AHR signaling: complex interactions involving the AHR repressor Involvement of Blimp-1 and AP-1 dysregulation in the 2,3,7,8-Tetrachlorodibenzo-p-dioxin-mediated suppression of the IgM response by B cells The aryl hydrocarbon receptor cross-talks with multiple signal transduction pathways Dynamic zebrafish interactome reveals transcriptional mechanisms of dioxin toxicity Expression of dioxin-related transactivating factors and target genes in human eutopic endometrial and endometriotic tissues Dioxin-mediated tumor progression through activation of mitochondria-to-nucleus stress signaling Aryl hydrocarbon receptor (AhR) agonists suppress interleukin-6 expression by bone marrow stromal cells: an immunotoxicology study The intersection between the aryl hydrocarbon receptor (AhR)- and retinoic acid-signaling pathways Aryl hydrocarbon receptor activation attenuates Per1 gene induction and influences circadian clock resetting Mechanisms of gender-specific TCDD-induced toxicity in guinea pig adipose tissue Activation of mitogen-activated protein kinases (MAPKs) by aromatic hydrocarbons: role in the regulation of aryl hydrocarbon receptor (AHR) function Aryl hydrocarbon receptor-dependent apoptotic cell death induced by the flavonoid chrysin in human colorectal cancer cells Activation of transcription factors activator protein-1 and nuclear factor-kappaB by 2,3,7,8-tetrachlorodibenzo-p-dioxin A mutation in the aryl hydrocarbon receptor (AHR) in a cultured mammalian cell line identifies a novel region of AHR that affects DNA binding A role of aryl hydrocarbon receptor in the antiandrogenic effects of polycyclic aromatic hydrocarbons in LNCaP human prostate carcinoma cells 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD)-induced activation of mitogen-activated protein kinase signaling pathway in Jurkat T cells Mechanisms of RAS/β-catenin interactions Perinatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin alters activity-dependent expression of BDNF mRNA in the neocortex and male rat sexual behavior in adulthood Role of the aryl hydrocarbon receptor in tobacco smoke extract-induced matrix metalloproteinase-1 expression. TCDD deregulates contact inhibition in rat liver oval cells via Ah receptor, JunD and cyclin A Early Life Exposure to Low Levels of AHR Agonist PCB126 (3,3',4,4',5-Pentachlorobiphenyl) Reprograms Gene Expression in Adult Brain. Nuclear receptors in the cross-talk of drug metabolism and inflammation. Regulation of cytochrome P4501B1 (CYP1B1) in mouse embryo fibroblast (C3H10T1/2) cells by protein kinase C (PKC)

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