About: Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis

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rdf:type	Item
description	artículu científicu espublizáu en 1999 (ast) vědecký článek publikovaný v roce 1999 (cs) مقالة علمية (نشرت في أبريل 1999) (ar) 1999 թվականի ապրիլին հրատարակված գիտական հոդված (hy) vedecký článok (publikovaný 1999/04/01) (sk) 1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած (hyw) наукова стаття, опублікована у квітні 1999 (uk) scientific journal article (en) im April 1999 veröffentlichter wissenschaftlicher Artikel (de) wetenschappelijk artikel (gepubliceerd op 1999/04/01) (nl)
publication date	wds:Q28585138-DF275C0B-AC00-4DB6-B6B7-ADEC6DD7877A
publication date	1999-04-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28585138-391F22B1-1245-44FB-B4AF-7EDC87C46B72
language of work or name	English
author name string	wds:Q28585138-12CE138D-CC75-4495-B744-225DF7C4FA81 wds:Q28585138-478C7D66-D920-442A-A3FD-435C06E3F4CA wds:Q28585138-545EF642-3849-41C8-A602-7754F43CC4DF wds:Q28585138-6502A657-AF96-4EC8-9579-C7FD83234A39 wds:Q28585138-7ECCEF74-DB81-4FD5-B37F-755BE95EB326 wds:Q28585138-87B8FF81-9AA1-4389-B465-E1842F23E7FE wds:Q28585138-B4D7E423-5EAA-4E25-9E1B-6F43F6613679 wds:Q28585138-E49A507F-A493-4D1F-9E90-7813866CB349 wds:Q28585138-E6CA23EC-5F75-45A5-9E84-079F6F266954
OpenCitations bibliographic resource ID	wds:Q28585138-F46302E1-785B-44ED-ACDF-10FC4483D2E0
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/5036977
author name string	P. E. Swanson C. M. Knudson K. C. Chang J. P. Cobb R. S. Hotchkiss I. E. Karl T. G. Buchman D. F. Osborne K. M. Zollner
OpenCitations bibliographic resource ID	5036977
rdfs:label	Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (en) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (nl) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (ast)
skos:prefLabel	Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (en) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (nl) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (ast)
name	Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (en) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (nl) Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (ast)
author	wds:Q28585138-120E71CE-B151-49E3-9009-4D216E22C1F4
author	Stanley J. Korsmeyer
title	wds:Q28585138-222ED451-856F-44DF-B0C4-E8451B18CAAA
title	Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis (en)
page(s)	wds:Q28585138-68E1D244-0312-4E2F-AFE8-4DF2204E1307
page(s)	4148–4156
instance of	wds:Q28585138-E1897405-3F76-44DE-AF4C-01356C714A3C
instance of	scholarly article
main subject	wds:Q28585138-0D36FF5B-EB87-4B54-85F9-62CDE34683F0 wds:Q28585138-AF8CEBD3-96D0-43EC-9396-58DE4B5E19F2 wds:Q28585138-D81DCCF1-A864-4B2F-AA41-78671A1A5BAB wds:Q28585138-EE24A024-EF0B-4903-A698-63287FE7B862
main subject	wd:Q14905404 sepsis apoptotic process overexpression
PubMed ID	wds:Q28585138-2D3872C4-609B-4229-8B10-188562688AD2
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/10201940
PubMed ID	10201940
published in	wds:Q28585138-829E1723-3923-42E5-A071-D91EA35C503F
published in	Journal of Immunology
issue	wds:Q28585138-973A5332-ED28-44DF-B7CD-F11CB9E5536C
volume	wds:Q28585138-75DEFC01-ADDF-4484-89DA-A85FCDA486C0
issue	7
volume	162
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28585138
is cites work of	Use of intracellular cytokine staining and bacterial superantigen to document suppression of the adaptive immune system in injured patients. Molecular Diagnostics in Sepsis: From Bedside to Bench Differential efficacy of caspase inhibitors on apoptosis markers during sepsis in rats and implication for fractional inhibition requirements for therapeutics CD8+ T cells promote inflammation and apoptosis in the liver after sepsis: role of Fas-FasL. A potential therapeutic role for P2X7 receptor (P2X7R) antagonists in the treatment of inflammatory diseases Mechanisms of immune resolution Thermal injury-plus-sepsis contributes to a substantial deletion of intestinal mesenteric lymph node CD4 T cell via apoptosis Mechanisms and regulation of the gene-expression response to sepsis. Sepsis-induced apoptosis leads to active suppression of delayed-type hypersensitivity by CD8+ regulatory T cells through a TRAIL-dependent mechanism Prevention of lymphocyte cell death in sepsis improves survival in mice Adoptive transfer of apoptotic splenocytes worsens survival, whereas adoptive transfer of necrotic splenocytes improves survival in sepsis PD-L1 blockade improves survival in experimental sepsis by inhibiting lymphocyte apoptosis and reversing monocyte dysfunction Upregulation of programmed death-1 on T cells and programmed death ligand-1 on monocytes in septic shock patients Caspase-1 regulates Escherichia coli sepsis and splenic B cell apoptosis independently of interleukin-1beta and interleukin-18 Cancer causes increased mortality and is associated with altered apoptosis in murine sepsis. CD4+ lymphocytes control gut epithelial apoptosis and mediate survival in sepsis Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy B and T lymphocyte attenuator expression on CD4+ T-cells associates with sepsis and subsequent infections in ICU patients Adoptive transfer of dying cells causes bystander-induced apoptosis Adenovirus-mediated gene expression in vivo is enhanced by the antiapoptotic bcl-2 gene The apoptotic pathway as a therapeutic target in sepsis Caspase-7 deficiency protects from endotoxin-induced lymphocyte apoptosis and improves survival. MyD88-dependent expansion of an immature GR-1(+)CD11b(+) population induces T cell suppression and Th2 polarization in sepsis Intestinal crosstalk: a new paradigm for understanding the gut as the "motor" of critical illness Immunosuppression in sepsis: a novel understanding of the disorder and a new therapeutic approach Genome-level longitudinal expression of signaling pathways and gene networks in pediatric septic shock The enigma of sepsis Preclinical evaluation of targeting the Nrf2 pathway by triterpenoids (CDDO-Im and CDDO-Me) for protection from LPS-induced inflammatory response and reactive oxygen species in human peripheral blood mononuclear cells and neutrophils Effect of Regulatory T Cells on Promoting Apoptosis of T Lymphocyte and Its Regulatory Mechanism in Sepsis

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