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| - artículu científicu espublizáu en 2000 (ast)
- vědecký článek publikovaný v roce 2000 (cs)
- наукова стаття, опублікована в липні 2000 (uk)
- 2000 թվականի հուլիսին հրատարակված գիտական հոդված (hy)
- 2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած (hyw)
- مقالة علمية (نشرت في يوليو 2000) (ar)
- vedecký článok (publikovaný 2000/07/01) (sk)
- wetenschappelijk artikel (gepubliceerd op 2000/07/01) (nl)
- scientific journal article (en)
- article scientifique publié en juillet 2000 (fr)
- im Juli 2000 veröffentlichter wissenschaftlicher Artikel (de)
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| OpenCitations bibliographic resource ID
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| author name string
| - N. Suzuki
- F. Hakuno
- H. Sakamoto
- K. Umesaki
- M. Akahori
- M. Ariga
- S. I. Takahashi
- T. Nedachi
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| - Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (en)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (nl)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (ast)
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| skos:prefLabel
| - Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (en)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (nl)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (ast)
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| name
| - Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (en)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (nl)
- Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (ast)
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| title
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| title
| - Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells (en)
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| DOI
| - 10.1210/EN.141.7.2429
- 10.1210/ENDO.141.7.7539
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| is about
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| is cites work
of | - Mechanism by which cAMP activates PI3-kinase and increases bile acid secretion in WIF-B9 cells
- Phosphatidylinositol 3-kinase-binding protein, PI3KAP/XB130, is required for cAMP-induced amplification of IGF mitogenic activity in FRTL-5 thyroid cells
- The association between thyroid autoantibodies in serum and abnormal function and structure of the thyroid.
- Regulation of the phosphatidylinositol 3-kinase, Akt/protein kinase B, FRAP/mammalian target of rapamycin, and ribosomal S6 kinase 1 signaling pathways by thyroid-stimulating hormone (TSH) and stimulating type TSH receptor antibodies in the thyroid
- 53BP2S, interacting with insulin receptor substrates, modulates insulin signaling
- Identification of microRNAs that mediate thyroid cell growth induced by TSH
- TSH signaling and cell survival in 3T3-L1 preadipocytes
- Phosphatidylinositol 3-kinase (PI3K) activity bound to insulin-like growth factor-I (IGF-I) receptor, which is continuously sustained by IGF-I stimulation, is required for IGF-I-induced cell proliferation.
- Growth hormone inhibition of glucose uptake in adipocytes occurs without affecting GLUT4 translocation through an insulin receptor substrate-2-phosphatidylinositol 3-kinase-dependent pathway
- Phosphatidylinositol 3-Kinase-Associated Protein (PI3KAP)/XB130 Crosslinks Actin Filaments through Its Actin Binding and Multimerization Properties In Vitro and Enhances Endocytosis in HEK293 Cells
- Tumor necrosis factor (TNF)-α-induced repression of GKAP42 protein levels through cGMP-dependent kinase (cGK)-Iα causes insulin resistance in 3T3-L1 adipocytes
- Effects of tyrosine kinase inhibitors on cell death induced by sodium fluoride and pertussis toxin in the pancreatic beta-cell line, RINm5F
- Paraquat-induced oxidative stress represses phosphatidylinositol 3-kinase activities leading to impaired glucose uptake in 3T3-L1 adipocytes.
- ERK phosphorylation is not increased in papillary thyroid carcinomas with BRAF(V600E) mutation compared to that of corresponding normal thyroid tissues
- The Emerging Roles of Thyroglobulin
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