About: Periostin as a novel factor responsible for ventricular dilation

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scientific journal article

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rdf:type	Item
description	article scientifique publié en 2004 (fr) artículu científicu espublizáu en 2004 (ast) vědecký článek publikovaný v roce 2004 (cs) 2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована у вересні 2004 (uk) 2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած (hyw) wetenschappelijk artikel (gepubliceerd op 2004/09/28) (nl) vedecký článok (publikovaný 2004/09/28) (sk) مقالة علمية (نشرت في 28-9-2004) (ar) scientific journal article (en) im September 2004 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q28574950-CBCA3D7E-7EBD-48C8-9A80-89193A6CC8E8
publication date	2004-09-28 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28574950-8D12DEAA-8080-4512-9C9D-B01FA60BF475
language of work or name	English
author name string	wds:Q28574950-0B88C7FF-E8C8-4148-AC4B-35E4B1EE52C3 wds:Q28574950-53292455-8B02-4AE1-8B51-4B5F275779E0 wds:Q28574950-6070DF0D-1B0B-4245-832E-95F5E6362FDC wds:Q28574950-67CB4A66-3B85-4549-B88B-171EA2A7F22C wds:Q28574950-7693AEE8-66B1-4D99-A4E2-14020D3CDDEC wds:Q28574950-81821E25-E726-48E6-A0B4-BDE3041E510F wds:Q28574950-934C83B3-6D2F-4F23-BB16-85D4268BF20F wds:Q28574950-B0A18677-2643-4929-86DD-3038EB704CD5 wds:Q28574950-C88F6D5D-25F1-4949-9239-6980AEEA8C34 wds:Q28574950-D4A282EE-8AB9-4532-89E8-94D43B77C386
author name string	Toshio Ogihara Ryuichi Morishita Yasuhiro Hasegawa Kayoko Kawashima Keijiro Sugimura Naruto Katsuragi Noriko Nakamura Tayehito Ochiai Yasufumi Kaneda Yoshiaki Taniyama
rdfs:label	Periostin as a novel factor responsible for ventricular dilation (en) Periostin as a novel factor responsible for ventricular dilation (nl) Periostin as a novel factor responsible for ventricular dilation (ast)
skos:prefLabel	Periostin as a novel factor responsible for ventricular dilation (en) Periostin as a novel factor responsible for ventricular dilation (nl) Periostin as a novel factor responsible for ventricular dilation (ast)
name	Periostin as a novel factor responsible for ventricular dilation (en) Periostin as a novel factor responsible for ventricular dilation (nl) Periostin as a novel factor responsible for ventricular dilation (ast)
title	wds:Q28574950-CD25A8D2-C352-4E11-A87C-B919C643ADCB
title	Periostin as a novel factor responsible for ventricular dilation (en)
page(s)	wds:Q28574950-C1B3F929-DE4D-4696-8AFA-BC66AF573979
page(s)	1806–1813
instance of	wds:Q28574950-3ACA02EC-BD27-47F5-BD1D-7CC9AFEF5FBF
instance of	scholarly article
PubMed ID	wds:Q28574950-6B240AFB-538D-4EFC-AC93-119D6F820EB5
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15381649
PubMed ID	15381649
published in	wds:Q28574950-E28D168D-7E84-4573-B6E6-CABAF7821D9F
published in	Circulation
issue	wds:Q28574950-9A0955BF-99E8-49A5-8C0D-C02491096715
volume	wds:Q28574950-BF466996-9686-425D-82B0-64C8F6658F93
issue	13
volume	110
DOI	wds:Q28574950-B3576A21-3CFC-47A6-A4AD-9AE3DCF343C3
DOI	http://dx.doi.org/10.1161/01.CIR.0000142607.33398.54
DOI	10.1161/01.CIR.0000142607.33398.54
ResearchGate publication ID	wds:Q28574950-10CCFF87-9B03-499E-A157-9AF5DE71683A
ResearchGate publication ID	8332623
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28574950
is cites work of	Periostin regulates atrioventricular valve maturation. Periostin promotes atrioventricular mesenchyme matrix invasion and remodeling mediated by integrin signaling through Rho/PI 3-kinase Metastatic outgrowth encompasses COL-I, FN1, and POSTN up-regulation and assembly to fibrillar networks regulating cell adhesion, migration, and growth Genetic enhancement of stem cell engraftment, survival, and efficacy Deconstructing fibrosis research: do pro-fibrotic signals point the way for chronic dermal wound regeneration? Periostin facilitates eosinophil tissue infiltration in allergic lung and esophageal responses Molecular and physiological characterization of RV remodeling in a murine model of pulmonary stenosis periostin null mice exhibit dwarfism, incisor enamel defects, and an early-onset periodontal disease-like phenotype Periostin advances atherosclerotic and rheumatic cardiac valve degeneration by inducing angiogenesis and MMP production in humans and rodents. Periostin regulates collagen fibrillogenesis and the biomechanical properties of connective tissues Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling Periostin-like-factor and Periostin in an animal model of work-related musculoskeletal disorder Recruitment of new cells into the postnatal heart: potential modification of phenotype by periostin Epicardial-derived cell epithelial-to-mesenchymal transition and fate specification require PDGF receptor signaling. Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing mice Cardiac Fibrosis: The Fibroblast Awakens Origin, development, and differentiation of cardiac fibroblasts Periostin is required for maturation and extracellular matrix stabilization of noncardiomyocyte lineages of the heart Stemming heart failure with cardiac- or reprogrammed-stem cells The AP-1 transcription factor c-Jun prevents stress-imposed maladaptive remodeling of the heart Ablation of periostin inhibits post-infarction myocardial regeneration in neonatal mice mediated by the phosphatidylinositol 3 kinase/glycogen synthase kinase 3β/cyclin D1 signalling pathway Genetic manipulation of periostin expression in the heart does not affect myocyte content, cell cycle activity, or cardiac repair P2Y6 receptor-Galpha12/13 signalling in cardiomyocytes triggers pressure overload-induced cardiac fibrosis Periostin is essential for cardiac healing after acute myocardial infarction Phosphatidylinositol-3-kinase signaling mediates vascular smooth muscle cell expression of periostin in vivo and in vitro Matricellular proteins in cardiac adaptation and disease The extracellular matrix: at the center of it all. Periostin induces proliferation of differentiated cardiomyocytes and promotes cardiac repair. Deletion of periostin reduces muscular dystrophy and fibrosis in mice by modulating the transforming growth factor-β pathway IL-6 loss causes ventricular dysfunction, fibrosis, reduced capillary density, and dramatically alters the cell populations of the developing and adult heart Periostin expression and epithelial-mesenchymal transition in cancer: a review and an update Angiotensin II increases periostin expression via Ras/p38 MAPK/CREB and ERK1/2/TGF-β1 pathways in cardiac fibroblasts Periostin promotes a fibroblastic lineage pathway in atrioventricular valve progenitor cells Periostin as a heterofunctional regulator of cardiac development and disease

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