About: The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms

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About: The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms Goto Sponge NotDistinct Permalink

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scientific journal article

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rdf:type	Item
description	article scientifique publié en 2008 (fr) artículu científicu espublizáu en 2009 (ast) vědecký článek publikovaný v roce 2009 (cs) 2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած (hyw) 2009 թվականի փետրվարին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в лютому 2009 (uk) مقالة علمية (نشرت في فبراير 2009) (ar) vedecký článok (publikovaný 2009/02/01) (sk) wetenschappelijk artikel (gepubliceerd op 2009/02/01) (nl) im Februar 2009 veröffentlichter wissenschaftlicher Artikel (de) scientific journal article (en)
publication date	wds:Q28574532-2C8169F8-DEE1-458D-95ED-8C4F68C090FA
publication date	2008-09-24 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28574532-AD707D89-436C-4064-9E37-C97C7CE2EFAC
language of work or name	English
cites work	wds:Q28574532-C58308C1-12C3-4E80-AA19-9185778A65B2
cites work	Redox regulation of 7-ketocholesterol-induced apoptosis by beta-carotene in human macrophages
author name string	wds:Q28574532-0FC849B6-89AB-43CA-B15F-9CC59F990720 wds:Q28574532-33782FE5-B489-437F-93B9-88FBC85CCCEA wds:Q28574532-AA77DFB9-E952-4980-9D63-82966FFC6D3C
OpenCitations bibliographic resource ID	wds:Q28574532-C3E2B34E-3972-4D8D-84AB-4D99369408F4
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/3597040
author name string	Irene Carmona-Cuenca Laia Caja Miguel M Murillo
OpenCitations bibliographic resource ID	3597040
rdfs:label	The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (en) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (nl) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (ast)
skos:prefLabel	The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (en) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (nl) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (ast)
name	The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (en) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (nl) The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (ast)
author	wds:Q28574532-0C796EB9-D356-47CA-BE37-2ACE3D17FAC9 wds:Q28574532-2F89C486-E071-4C59-9919-C9498C813B0F wds:Q28574532-83E829AF-DF01-4EBF-8074-A74F4087CD5B
author	Isabel Fabregat Patricia Sancho Esther Bertran
title	wds:Q28574532-214CA56C-D85C-40E6-84B4-83090BEED0D0
title	The inhibition of the epidermal growth factor (EGF) pathway enhances TGF-beta-induced apoptosis in rat hepatoma cells through inducing oxidative stress coincident with a change in the expression pattern of the NADPH oxidases (NOX) isoforms (en)
page(s)	wds:Q28574532-B7F773A3-9235-4F52-989D-63732EDEAA05
page(s)	253-263
instance of	wds:Q28574532-096091B3-0AC7-4998-99C4-F49B5E52E8ED
instance of	scholarly article
main subject	wds:Q28574532-0D6C6E06-0C36-4637-A021-AC78787705FD wds:Q28574532-5C0FCD57-D3BB-4076-8911-AF98650FAD58 wds:Q28574532-F27A9E56-0A3C-4552-878B-75F874027050
main subject	hepatocellular carcinoma apoptotic process nitrogen oxide
PubMed ID	wds:Q28574532-F1B10B7E-8EBB-4E27-BDED-88FC5573A72F
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/18848961
PubMed ID	18848961
published in	wds:Q28574532-EFFFA5E9-6A2F-4202-BAAE-3BB80C81D331
published in	Biochimica et Biophysica Acta
issue	wds:Q28574532-978B80DF-926B-43E4-99DE-A8FE25CC9D1A
volume	wds:Q28574532-DFCA5DEC-AF9C-4D37-A576-BA142908DBC1
issue	2
volume	1793
DOI	wds:Q28574532-A0CCE3D9-B3F6-41C8-AA6C-1ABD166CF2BB
DOI	http://dx.doi.org/10.1016/J.BBAMCR.2008.09.003
DOI	10.1016/J.BBAMCR.2008.09.003
ResearchGate publication ID	wds:Q28574532-3711BD0B-47E5-44CC-840E-995C3A276B7F
ResearchGate publication ID	23311883
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28574532
is cites work of	Role of NADPH oxidases in the redox biology of liver fibrosis Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system The EGFR signalling system in the liver: from hepatoprotection to hepatocarcinogenesis Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Oxidative stress and glutathione in TGF-beta-mediated fibrogenesis Oxidative modification of nuclear mitogen-activated protein kinase phosphatase 1 is involved in transforming growth factor beta1-induced expression of plasminogen activator inhibitor 1 in fibroblasts. Liver fibrosis and hepatocyte apoptosis are attenuated by GKT137831, a novel NOX4/NOX1 inhibitor in vivo. NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development. TGF-β regulates Nox4, MnSOD and catalase expression, and IL-6 release in airway smooth muscle cells. Nox4 involvement in TGF-beta and SMAD3-driven induction of the epithelial-to-mesenchymal transition and migration of breast epithelial cells. Protein-tyrosine phosphatase 1B (PTP1B) deficiency confers resistance to transforming growth factor-β (TGF-β)-induced suppressor effects in hepatocytes Signaling mechanism(s) of reactive oxygen species in Epithelial-Mesenchymal Transition reminiscent of cancer stem cells in tumor progression The NOX toolbox: validating the role of NADPH oxidases in physiology and disease Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice Oxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma Oxidative stress and pulmonary fibrosis NADPH Oxidases NOXs and DUOXs as putative targets for cancer therapy. Mouse hepatic oval cells require Met-dependent PI3K to impair TGF-β-induced oxidative stress and apoptosis The prognostic significance of growth factors and growth factor receptors in gastric adenocarcinoma. Caveolin-1 is required for TGF-β-induced transactivation of the EGF receptor pathway in hepatocytes through the activation of the metalloprotease TACE/ADAM17. Indole-3- carbinol enhances sorafenib cytotoxicity in hepatocellular carcinoma cells: A mechanistic study NADPH oxidase NOX1 controls autocrine growth of liver tumor cells through up-regulation of the epidermal growth factor receptor pathway European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS). NADPH oxidase biology and the regulation of tyrosine kinase receptor signaling and cancer drug cytotoxicity Caveolin-1-dependent activation of the metalloprotease TACE/ADAM17 by TGF-β in hepatocytes requires activation of Src and the NADPH oxidase NOX1. Liver inflammation and fibrosis The level of caveolin-1 expression determines response to TGF-β as a tumour suppressor in hepatocellular carcinoma cells. Snail mediates crosstalk between TGFβ and LXRα in hepatocellular carcinoma Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis. Bergenin decreases the morphine-induced physical dependence via antioxidative activity in mice. Intrahepatic microcirculatory disorder, parenchymal hypoxia and NOX4 upregulation result in zonal differences in hepatocyte apoptosis following lipopolysaccharide- and D-galactosamine-induced acute liver failure in rats

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