About: Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma

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scientific journal article

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rdf:type	Item
description	artículu científicu espublizáu en 1997 (ast) vědecký článek publikovaný v roce 1997 (cs) 1997 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած (hyw) 1997 թվականի հոտեմբերին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в жовтні 1997 (uk) article publié dans la revue scientifique Science (fr) wetenschappelijk artikel (gepubliceerd op 1997/10/10) (nl) vedecký článok (publikovaný 1997/10/10) (sk) научни чланак (објављен 1997/10/10) (sr) مقالة علمية (نشرت في 10-10-1997) (ar) scientific journal article (en) im Oktober 1997 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q28512432-93F02782-1DB2-472A-92A7-86A1DF2922AB
publication date	1997-10-10 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28512432-B2CBBA5F-9446-424A-9D3E-9D2BC88FB01F
language of work or name	English
author name string	wds:Q28512432-139B1226-8225-467F-8C9E-381B8876BEF0 wds:Q28512432-2D6F6F0A-F169-4D35-A2B8-0080DC134A1B wds:Q28512432-69E40467-07D0-4A03-96C7-F5F446CE6B83 wds:Q28512432-82F8F92A-5084-4338-B4F7-64EF7D6B172E
OpenCitations bibliographic resource ID	wds:Q28512432-6E6FA250-509F-4712-B92D-6B781BF32B68
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/2577549
author name string	C. Chen S. Tonegawa A. B. Malmberg A. I. Basbaum
OpenCitations bibliographic resource ID	2577549
rdfs:label	Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (en) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (nl) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (ast)
skos:prefLabel	Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (en) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (nl) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (ast)
name	Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (en) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (nl) Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (ast)
title	wds:Q28512432-5A9EBEA3-CE9E-4EE0-B2A6-D111BD023C56
title	Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma (en)
page(s)	wds:Q28512432-F89FD725-C4E4-4BC3-89AA-2FF6579A3908
page(s)	279–283
instance of	wds:Q28512432-AD6F9B6E-133F-43D6-9303-95E116C54958
instance of	scholarly article
main subject	wds:Q28512432-85BFE611-A162-41F9-8D5C-DBD2C78AE8E9 wds:Q28512432-E2A95E86-BF00-49BC-85AC-056526AEAD28
main subject	neuropathic pain Protein kinase C, gamma
PubMed ID	wds:Q28512432-F524AD6C-8A98-40A0-86AA-FD8A5B616B95
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/9323205
PubMed ID	9323205
published in	wds:Q28512432-E2E85682-9BD9-4501-8C97-EC08E2353B07
published in	Science
issue	wds:Q28512432-C28956DD-8638-40F3-B102-2354F993DE46
volume	wds:Q28512432-E16FF436-E7F6-4BDC-823A-C628F797A93F
issue	5336
volume	278
DOI	wds:Q28512432-D3153B40-196D-45BE-B7A8-307CE4452B86
DOI	http://dx.doi.org/10.1126/SCIENCE.278.5336.279
DOI	10.1126/SCIENCE.278.5336.279
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28512432
is cites work of	Descending control of pain Transmitting pain and itch messages: a contemporary view of the spinal cord circuits that generate gate control. The science of migraine Alteration in the voltage dependence of NMDA receptor channels in rat dorsal horn neurones following peripheral inflammation Anti-allodynic action of the tormentic acid, a triterpene isolated from plant, against neuropathic and inflammatory persistent pain in mice. Protease-activated receptor 2 sensitizes the capsaicin receptor transient receptor potential vanilloid receptor 1 to induce hyperalgesia. Specific Involvement of PKC-ε in Sensitization of the Neuronal Response to Painful Heat Potentiation of glutamatergic synaptic transmission by protein kinase C-mediated sensitization of TRPV1 at the first sensory synapse L1 cell adhesion molecule is essential for the maintenance of hyperalgesia after spinal cord injury. VGLUT2 expression in primary afferent neurons is essential for normal acute pain and injury-induced heat hypersensitivity. Lesion of the dopaminergic nigrostriatal pathway induces trigeminal dynamic mechanical allodynia Inflammation-induced up-regulation of protein kinase Cgamma immunoreactivity in rat spinal cord correlates with enhanced nociceptive processing. Reduced hyperalgesia induced by nerve injury, but not by inflammation in mice lacking protein kinase C gamma isoform Loose ligation of the rat sciatic nerve is accompanied by changes in the subcellular content of protein kinase C beta II and gamma in the spinal dorsal horn PKCgamma contributes to a subset of the NMDA-dependent spinal circuits that underlie injury-induced persistent pain. M-CSF induced differentiation of myeloid precursor cells involves activation of PKC-delta and expression of Pkare The induction of pain: an integrative review. Increased phosphorylation of the GluR1 subunit of spinal cord α-amino-3-hydroxy-5-methyl-4-isoxazole propionate receptor in rats following intradermal injection of capsaicin Primary afferent tachykinins are required to experience moderate to intense pain The sensory neurons of touch Injury-induced mechanical hypersensitivity requires C-low threshold mechanoreceptors The molecular dynamics of pain control Enhanced mu-opioid responses in the spinal cord of mice lacking protein kinase Cgamma isoform Spinal G-protein-gated potassium channels contribute in a dose-dependent manner to the analgesic effect of mu- and delta- but not kappa-opioids. Pathophysiological tissue changes associated with repetitive movement: a review of the evidence Craniofacial pain and motor function: pathogenesis, clinical correlates, and implications Cell signaling and the genesis of neuropathic pain Venom from the platypus, Ornithorhynchus anatinus, induces a calcium-dependent current in cultured dorsal root ganglion cells Glutamate receptors and nociception: implications for the drug treatment of pain Antinociceptive action of the extract and the flavonoid quercitrin isolated from Bauhinia microstachya leaves Protein Kinase Cα Negatively Regulates Systolic and Diastolic Function in Pathological Hypertrophy The spinal biology in humans and animals of pain states generated by persistent small afferent input Expression of calcitonin gene-related peptide, substance P and protein kinase C in cultured dorsal root ganglion neurons following chronic exposure to mu, delta and kappa opiates Protease-activated receptor-1 and platelet-derived growth factor in spinal cord neurons are implicated in neuropathic pain after nerve injury The TIP39-PTH2 receptor system: unique peptidergic cell groups in the brainstem and their interactions with central regulatory mechanisms Anatomical and physiological evidence for involvement of tuberoinfundibular peptide of 39 residues in nociception. A diverse family of GPCRs expressed in specific subsets of nociceptive sensory neurons Distribution of antinociceptive adenosine A1 receptors in the spinal cord dorsal horn, and relationship to primary afferents and neuronal subpopulations Initiation of neuropathic pain requires lysophosphatidic acid receptor signaling Phosphorylation of CREB and mechanical hyperalgesia is reversed by blockade of the cAMP pathway in a time-dependent manner after repeated intramuscular acid injections.

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