About: Christopher Shaw

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Professor of Neurology and Neurogenetics

Attributes	Values
rdf:type	Item
description	ikertzailea (eu) 연구원 (ko) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) Professor of Neurology and Neurogenetics (en)
rdfs:label	Christopher Shaw (en) ক্রিস্টোফার শ (bn) Christopher Shaw (es) Christopher Shaw (nl) Christopher Shaw (ast)
skos:prefLabel	Christopher Shaw (en) ক্রিস্টোফার শ (bn) Christopher Shaw (es) Christopher Shaw (nl) Christopher Shaw (ast)
name	Christopher Shaw (en) ক্রিস্টোফার শ (bn) Christopher Shaw (es) Christopher Shaw (nl) Christopher Shaw (ast)
given name	wds:Q24005568-4DCCD63B-ED0E-48CD-B1D8-103F36085148
given name	Christopher
instance of	wds:Q24005568-4352BD7C-D204-4D02-957E-B3446AD5E371
instance of	Homo sapiens
award received	wds:Q24005568-5876E879-9C10-4789-8E03-B074763C7152 wds:Q24005568-939E4ECE-26D3-4C24-A084-8226235E8821 wds:Q24005568-C9BBE591-9021-4DEF-B847-BB75803676CF
award received	Fellow of the Royal College of Physicians of London Fellow of the Academy of Medical Sciences Fellow of the Australian Academy of Health and Medical Sciences
Google Knowledge Graph ID	wds:Q24005568-F7D87A64-AF8C-43F5-BD25-B8523E4D16CD
Google Knowledge Graph ID	http://g.co/kg/g/11cmr0znng
Google Knowledge Graph ID	/g/11cmr0znng
Google Scholar author ID	wds:Q24005568-34d30935-4abe-676f-313c-9a852728d154
Google Scholar author ID	5pUNbawAAAAJ
skos:altLabel	Christopher E Shaw (en) Christopher E Shaw (nl) Christopher E Shaw (ast) Christopher Edward Dennistoun Shaw (en)
occupation	wds:Q24005568-74719D49-6E52-456F-999A-C730A5721BBB wds:Q24005568-855F4396-46FD-4BB0-B9FF-E1D066AA2A0B
employer	wds:Q24005568-BC12B697-E26A-4414-9520-24C8C63D0B82 wds:Q24005568-DD8DC1B3-6347-4E68-ADCC-AECADA9F0362
different from	wds:Q24005568-a8925a29-4902-2fb5-0ec9-d2ceada04c67
sex or gender	wds:Q24005568-DEA5557F-7723-4029-997F-7F51A72E35F2
member of	wds:Q24005568-ABF43284-0637-4276-8F13-23F1BF1369E6
Who's Who UK ID	wds:Q24005568-f02fe58b-44ed-f52b-045c-8fc8bdf72019
date of birth	wds:Q24005568-11A4208C-E061-4F20-B797-8352747E71AB
educated at	wds:Q24005568-19087B93-434C-40B7-8EB0-90EC9CCA0F91 wds:Q24005568-6397A117-B3A9-4076-B515-59B310D0B21F wds:Q24005568-A08ECD98-755D-4807-A939-FE6F69EB66D8 wds:Q24005568-A0B12DB6-9325-4D4A-B5D6-0956EBA8328D wds:Q24005568-F123E4D8-67D6-451F-A347-D34CB65DE970
family name	wds:Q24005568-93AC5B2C-A68F-40C3-B1A7-C9BBE123AF89
occupation	neurologist researcher
employer	King's College London
different from	Christopher Shaw
sex or gender	male
member of	Royal College of Physicians, London
Who's Who UK ID	U245495
date of birth	1960-01-01 00:00:00Z (xsd:dateTime)
educated at	Royal College of Physicians, London Royal Australasian College of Physicians University of Otago
family name	Shaw
ORCID iD	wds:Q24005568-49F64765-6C25-4CA2-9BF1-FC7BFC33EF50
ORCID iD	https://orcid.org/0000-0003-0251-0146
ORCID iD	0000-0003-0251-0146
is owl:sameAs of	Christopher Shaw Christopher Shaw
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q24005568 Christopher Shaw (neurologist)
is author of	Volumetric cortical loss in sporadic and familial amyotrophic lateral sclerosis Amyotrophic lateral sclerosis-like superoxide dismutase 1 proteinopathy is associated with neuronal loss in Parkinson's disease brain D90A-SOD1 mediated amyotrophic lateral sclerosis: a single founder for all cases with evidence for a Cis-acting disease modifier in the recessive haplotype. Allele-specific knockdown of ALS-associated mutant TDP-43 in neural stem cells derived from induced pluripotent stem cells Human iPSC-derived motoneurons harbouring TARDBP or C9ORF72 ALS mutations are dysfunctional despite maintaining viability Age of onset of amyotrophic lateral sclerosis is modulated by a locus on 1p34.1. TUBA4A gene analysis in sporadic amyotrophic lateral sclerosis: identification of novel mutations. TDP-43 Proteinopathy and ALS: Insights into Disease Mechanisms and Therapeutic Targets Mutations in the vesicular trafficking protein annexin A11 are associated with amyotrophic lateral sclerosis. Ciliary neurotrophic factor genotype does not influence clinical phenotype in amyotrophic lateral sclerosis C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicity. C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicity. Mitochondrial abnormalities and disruption of the neuromuscular junction precede the clinical phenotype and motor neuron loss in hFUSWT transgenic mice. H63D polymorphism in HFE is not associated with amyotrophic lateral sclerosis RNA Misprocessing in C9orf72-Linked Neurodegeneration Predicting the future of ALS: the impact of demographic change and potential new treatments on the prevalence of ALS in the United Kingdom, 2020-2116 Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease A feedback loop between dipeptide-repeat protein, TDP-43 and karyopherin-α mediates C9orf72-related neurodegeneration Objectively Monitoring Amyotrophic Lateral Sclerosis Patient Symptoms During Clinical Trials With Sensors: Observational Study ALS-specific cognitive and behavior changes associated with advancing disease stage in ALS

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