About: Marcello Pinti

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researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) वैज्ञानिक (hi) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	Marcello Pinti (en) Marcello Pinti (es) Marcello Pinti (it) Marcello Pinti (nl) Marcello Pinti (sl) Marcello Pinti (ast)
skos:prefLabel	Marcello Pinti (en) Marcello Pinti (es) Marcello Pinti (it) Marcello Pinti (nl) Marcello Pinti (sl) Marcello Pinti (ast)
name	Marcello Pinti (en) Marcello Pinti (es) Marcello Pinti (it) Marcello Pinti (nl) Marcello Pinti (sl) Marcello Pinti (ast)
Scopus author ID	wds:Q41701743-F8D218D3-2C86-4F49-9241-DE36B7DFEAB5
Scopus author ID	6701578776
given name	wds:Q41701743-CD9ABD64-44CD-4AC3-884A-5C6A4AAE3DCA
given name	Marcello
instance of	wds:Q41701743-8E40A98D-BD2E-40D4-889A-9EFEEF392D99
instance of	Homo sapiens
occupation	wds:Q41701743-42E6F19D-5F78-4BC9-BED3-F403B3C55C44
sex or gender	wds:Q41701743-EBBA1DEA-0930-4C04-93AE-6823A3046EBA
educated at	wds:Q41701743-0F38CA1D-D564-4C5B-8336-3D5D6A359985 wds:Q41701743-C822B021-6A52-47FA-848E-31305FCC9506
family name	wds:Q41701743-FD574A44-A91C-469A-9FCE-F788952F4EAD
occupation	researcher
sex or gender	male
educated at	University of Modena and Reggio Emilia University of Milan
family name	Pinti
ORCID iD	wds:Q41701743-36EAD7DF-175E-4756-9C84-7F56C3E0116D
ORCID iD	https://orcid.org/0000-0001-9118-1262
ORCID iD	0000-0001-9118-1262
IRIS UNIBO author ID	wds:Q41701743-044B16CF-9723-4FF6-8FDA-1CD35A8A7BC1
IRIS UNIBO author ID	185684
ResearcherID	wds:Q41701743-34C81612-B5D8-4FCA-8A04-15A6D4C13D79
Publons author ID	wds:Q41701743-795da2fc-f9dd-4ee9-9ded-76a032c12a9b
ResearcherID	C-9351-2015
Publons author ID	1232891
IRIS UNIMORE author ID	wds:Q41701743-B1B5FE41-BAAB-42DC-ADA8-61A7A18B35F1
IRIS UNIMORE author ID	05783
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q41701743
is author of	Mitochondrial Lon protease at the crossroads of oxidative stress, ageing and cancer. Early changes in intramitochondrial cardiolipin distribution during apoptosis. Natural Compounds Modulating Mitochondrial Functions Mitochondrial neurogastrointestinal encephalomyopathy: evidence of mitochondrial DNA depletion in the small intestine Multiparametric analysis of cells with different mitochondrial membrane potential during apoptosis by polychromatic flow cytometry Mitochondria hyperfusion and elevated autophagic activity are key mechanisms for cellular bioenergetic preservation in centenarians. Anti-HIV drugs and the mitochondria Mitochondrial liaisons of p53. Resistance of mtDNA-depleted cells to apoptosis Essential requirement of reduced glutathione (GSH) for the anti-oxidant effect of the flavonoid quercetin Mitochondrial DNA: a proinflammatory 'enemy from within' during HIV infection? Interfering with ROS Metabolism in Cancer Cells: The Potential Role of Quercetin. Quercetin inhibits lymphocyte activation and proliferation without inducing apoptosis in peripheral mononuclear cells Characterization of cells with different mitochondrial membrane potential during apoptosis Hepatoma HepG2 cells as a model for in vitro studies on mitochondrial toxicity of antiviral drugs: which correlation with the patient? Effects of the change from Stavudine to tenofovir in human immunodeficiency virus-infected children treated with highly active antiretroviral therapy: studies on mitochondrial toxicity and thymic function Protective effect of acetyl-l-carnitine against oxidative stress induced by antiretroviral drugs Mitochondrial alterations and tendency to apoptosis in peripheral blood cells from children with Down syndrome OPA1 mutations associated with dominant optic atrophy impair oxidative phosphorylation and mitochondrial fusion Mitochondrial heterogeneity during staurosporine-induced apoptosis in HL60 cells: Analysis at the single cell and single organelle level Simultaneous analysis of reactive oxygen species and reduced glutathione content in living cells by polychromatic flow cytometry Quercetin and cancer chemoprevention Subject classification obtained by cluster analysis and principal component analysis applied to flow cytometric data. Placenta and cord blood mitochondrial DNA toxicity in HIV-infected women receiving nucleoside reverse transcriptase inhibitors during pregnancy Aging with HIV infection: a journey to the center of inflammAIDS, immunosenescence and neuroHIV. Thymic output and functionality of the IL-7/IL-7 receptor system in centenarians: implications for the neolymphogenesis at the limit of human life. Cytometry, immunology, and HIV infection: three decades of strong interactions. Stable changes in CD4+ T lymphocyte miRNA expression after exposure to HIV-1 Altered clonogenic capability and stromal cell function characterize bone marrow of HIV-infected subjects with low CD4+ T cell counts despite viral suppression during HAART. Features of 'CD4-exploders', HIV-positive patients with an optimal immune reconstitution after potent antiretroviral therapy. Upregulation of nuclear-encoded mitochondrial LON protease in HAART-treated HIV-positive patients with lipodystrophy: implications for the pathogenesis of the disease Balanced regulation of mRNA production for Fas and Fas ligand in lymphocytes from centenarians: how the immune system starts its second century Direct analysis of thymic function in children with Down's syndrome Homeostatic cytokines and expansion of regulatory T cells accompany thymic impairment in children with Down syndrome. Genetic polymorphisms of Fas (CD95) and Fas ligand (CD178) influence the rise in CD4+ T cell count after antiretroviral therapy in drug-naïve HIV-positive patients Cytotoxic granule release dominates gag-specific CD4+ T-cell response in different phases of HIV infection Quality assessment of human mitochondrial DNA quantification: MITONAUTS, an international multicentre survey Increased mitochondrial DNA content in peripheral blood lymphocytes from HIV-infected patients with lipodystrophy Decreased apoptosis of bone marrow progenitor cells in HIV-1-infected patients during highly active antiretroviral therapy Increased plasma levels of extracellular mitochondrial DNA during HIV infection: A new role for mitochondrial damage-associated molecular patterns during inflammation

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