About: Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning

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description	wissenschaftlicher Artikel (de) article scientifique publié en 2008 (fr) artículu científicu espublizáu en 2008 (ast) наукова стаття, опублікована в липні 2008 (uk) 2008 թվականի հուլիսին հրատարակված գիտական հոդված (hy) 2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած (hyw) مقالة علمية (نشرت في يوليو 2008) (ar) wetenschappelijk artikel (gepubliceerd op 2008/07/01) (nl) scientific journal article (en)
publication date	wds:Q28771386-FFBC5EDC-14C4-4F16-BC90-CAC8047A3736
publication date	2008-07-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28771386-DD07F60A-2069-4940-ABC7-4AA865311552
language of work or name	English
cites work	wds:Q28771386-12493AF3-FEF3-4E08-8FC1-6B14ACB3E5F7 wds:Q28771386-392BD6AF-CA8A-404D-AA24-8A363AC2B837 wds:Q28771386-3A15CA9E-4564-4F98-80F6-89E5210CEEB9 wds:Q28771386-3FA272A9-1634-49B8-8457-34F63DDCBB23 wds:Q28771386-4647B4BE-DC75-411F-B45B-CCB5792C0323 wds:Q28771386-81ADF79A-CD27-4F92-A267-07300234F93C wds:Q28771386-8719D50A-966B-462D-B4C2-525B216CD462 wds:Q28771386-892DDF09-635D-4B27-910F-3DD802D12209 wds:Q28771386-91DD096C-EDA1-4A84-8578-A6952F89B461 wds:Q28771386-956E8B1B-065D-4D5A-97AE-19C8CC420427 wds:Q28771386-AC95C39B-30F6-4F05-837B-A7E1E1F94C2D wds:Q28771386-ACDEA8B7-7909-4D95-8B5E-44B51F3C7CF5 wds:Q28771386-BF350B5F-8116-43AD-9240-8D3C8ADC1F88 wds:Q28771386-BF7EBAA4-C3AB-4B36-8890-66BAB98BF323 wds:Q28771386-CD88E304-E2E8-4731-88CD-2BFBEB4E7D69 wds:Q28771386-E23A82D6-8671-4A88-9A42-3E52F4517754 wds:Q28771386-E2EA97F6-0E60-467C-AE09-FDF1ADEFFC26 wds:Q28771386-E3E26E20-4B44-408D-9F56-F1280F9446EC wds:Q28771386-FD013548-60D3-45EE-9C3E-BE5C6CB98826
cites work	STAT3 as an adapter to couple phosphatidylinositol 3-kinase to the IFNAR1 chain of the type I interferon receptor Ischemic preconditioning activates phosphatidylinositol-3-kinase upstream of protein kinase C Role of the JAK-STAT pathway in protection against myocardial ischemia/reperfusion injury Role of the JAK-STAT pathway in myocardial injury Tumor necrosis factor-induced protection of the murine heart is independent of p38-MAPK activation. Role of STAT3 in ischemic preconditioning PI3 kinase and not p42/p44 appears to be implicated in the protection conferred by ischemic preconditioning Identification of a novel role for sphingolipid signaling in TNF alpha and ischemic preconditioning mediated cardioprotection Classic ischemic but not pharmacologic preconditioning is abrogated following genetic ablation of the TNFalpha gene Cross-talk between the survival kinases during early reperfusion: its contribution to ischemic preconditioning Free radicals trigger TNF alpha-induced cardioprotection TNFalpha is required to confer protection in an in vivo model of classical ischaemic preconditioning TNFalpha-induced cytoprotection requires the production of free radicals within mitochondria in C2C12 myotubes Pharmacological preconditioning with tumor necrosis factor-alpha activates signal transducer and activator of transcription-3 at reperfusion without involving classic prosurvival kinases (Akt and extracellular signal-regulated kinase). Carbon monoxide differentially modulates STAT1 and STAT3 and inhibits apoptosis via a phosphatidylinositol 3-kinase/Akt and p38 kinase-dependent STAT3 pathway during anoxia-reoxygenation injury. New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway Survival kinases in ischemic preconditioning and postconditioning. Nuclear targeting of Akt enhances kinase activity and survival of cardiomyocytes. The JAK/STAT pathway is essential for opioid-induced cardioprotection: JAK2 as a mediator of STAT3, Akt, and GSK-3 beta
author name string	wds:Q28771386-1B7661FD-90F5-4AA4-AE7E-45C7E945F889 wds:Q28771386-3A22B60F-FE49-4315-833F-2D7139D48DC7 wds:Q28771386-72391426-7C50-401F-BD3E-9733D27D53B8 wds:Q28771386-76EFA39D-49F5-41C8-A41A-D65B1A57F011
OpenCitations bibliographic resource ID	wds:Q28771386-90536A7F-1F1A-4B30-A415-C282A3039355
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/2882785
author name string	Robert Smith Naushaad Suleman Sandrine C. Lecour Sarin Somers
OpenCitations bibliographic resource ID	2882785
rdfs:label	Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (en) Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (ast)
skos:prefLabel	Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (en) Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (ast)
name	Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (en) Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (ast)
author	wds:Q28771386-D4AB1469-1569-4B71-918F-8EA325533603
author	Lionel Opie
title	wds:Q28771386-A42869E5-026F-485E-AAD6-1FAC1ACE38EF
title	Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning (en)
page(s)	wds:Q28771386-51AE185C-278F-4814-B673-915D0AF397FE
page(s)	127–133
instance of	wds:Q28771386-A584E038-B87A-4DE9-B4FE-822BAEE0519A
instance of	scholarly article
PubMed ID	wds:Q28771386-AA0EC2DC-0B34-425F-B85D-88A205535481
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/18339648
PubMed ID	18339648
published in	wds:Q28771386-512BAF01-2610-465D-9CF0-ED4F6979D895
published in	Cardiovascular Research
issue	wds:Q28771386-87895A34-962E-4BC2-87BD-126CF79996DE
volume	wds:Q28771386-F7F9D072-D766-4520-84E2-D0F28858A328
issue	1
volume	79
DOI	wds:Q28771386-83B72D5C-56AD-486A-99F8-353DCA784EA4
DOI	http://dx.doi.org/10.1093/CVR/CVN067
DOI	10.1093/CVR/CVN067
ResearchGate publication ID	wds:Q28771386-BB903281-D1FC-493D-958E-C34A163382AB
ResearchGate publication ID	5511993
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28771386
is cites work of	Multiple protective pathways against reperfusion injury: a SAFE path without Aktion? Loss of the miR-144/451 cluster impairs ischaemic preconditioning-mediated cardioprotection by targeting Rac-1. Remote ischemic preconditioning confers late protection against myocardial ischemia-reperfusion injury in mice by upregulating interleukin-10. Exercise training preserves ischemic preconditioning in aged rat hearts by restoring the myocardial polyamine pool N-acetylcysteine and allopurinol synergistically enhance cardiac adiponectin content and reduce myocardial reperfusion injury in diabetic rats Signal transducer and activator of transcription 3 is involved in the cardioprotective signalling pathway activated by insulin therapy at reperfusion Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway The mitochondrial permeability transition pore and its role in myocardial ischemia reperfusion injury.

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