About: Calcium depletion dissociates and activates heterodimeric notch receptors

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description	artículu científicu espublizáu en 2000 (ast) vědecký článek publikovaný v roce 2000 (cs) wüsseschaftlicher Artikel, wo im März 2000 veröffentlicht worden isch (gsw) مقالة علمية (نشرت في مارس 2000) (ar) im März 2000 veröffentlichter wissenschaftlicher Artikel (de) 2000 թվականի մարտին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в березні 2000 (uk) 2000 թուականի Մարտին հրատարակուած գիտական յօդուած (hyw) vedecký článok (publikovaný 2000/03/01) (sk) wetenschappelijk artikel (gepubliceerd op 2000/03/01) (nl) article scientifique publié en 2000 (fr) scientific journal article (en)
publication date	wds:Q28609673-D7BB12AA-68C0-4001-811C-A0575D94BF89
publication date	2000-03-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28609673-9C3AC109-6A9F-41CD-881E-717E7D08CBCC
language of work or name	English
cites work	wds:Q28609673-12196EC9-C76B-44CE-9215-75EA53070992 wds:Q28609673-18DB7552-3D49-432C-8759-1A11834AEEB1 wds:Q28609673-234D7912-41B4-4343-A554-AD0C4F1D44C3 wds:Q28609673-264740F3-769D-4EE4-8573-703403B84DEB wds:Q28609673-28850C1D-5A38-4CF1-9191-FB986540B7E7 wds:Q28609673-2F70E74D-781F-4291-8A90-0BA76879048E wds:Q28609673-3681C8FF-FC5D-4638-8B76-C3B36A982A0A wds:Q28609673-36C64CD1-7132-46B2-82B6-E24DCA2A569E wds:Q28609673-3F4F3931-C6E8-4555-8DE9-731B0B88F93D wds:Q28609673-41D65CB4-31E8-43AE-8837-94950E97D12D wds:Q28609673-45DA4E30-E12D-4FA9-A834-ABE9D735FA8F wds:Q28609673-4863A2B2-AB69-4284-B9DC-70C12D0248A5 wds:Q28609673-4C63C1D5-CFC1-4CD8-8006-70DE35DB6C08 wds:Q28609673-4E40ACF8-43E5-4F14-9BE3-2C31C415AA15 wds:Q28609673-50CA80D9-F503-41BD-960E-3DF7D7BBFF3D wds:Q28609673-6244A9E4-6D8B-41C9-88FF-4836CFA7BCB6 wds:Q28609673-6930F32B-B6B5-4909-931E-27E0DB49DEB5 wds:Q28609673-732CD5F9-8245-428F-962D-8DDB6F50471F wds:Q28609673-74820223-1F99-43EE-B851-9577F19B2706 wds:Q28609673-81F8F51B-1330-4199-A50E-0FF699DA7AA8 wds:Q28609673-839A2C46-5384-4ABD-8837-F91A445C429E wds:Q28609673-87DCD1C0-A081-4AB7-B839-BD731BF75997 wds:Q28609673-88ACA1B6-BDE2-407F-8402-6D6468666986 wds:Q28609673-9CD100EC-8C8C-4E5C-A3B1-6F6420230DAD wds:Q28609673-A204AD39-4CCF-488B-98D6-E65B2920D527 wds:Q28609673-A4FADEA7-9A0F-4F91-B2A3-918F6E094467 wds:Q28609673-A87EA0B8-1923-4C22-B1B4-00155D2A6FA8 wds:Q28609673-B863A87E-E659-4E0D-8122-4DA86F166A31 wds:Q28609673-B88D1241-B4B1-4821-9302-5987ABECAE93 wds:Q28609673-BA45A031-CB1A-47BB-971C-C245CF6CCDE3 wds:Q28609673-BCE838A7-DC00-4F1D-B988-F17F71CB3F48 wds:Q28609673-C0205F05-9153-47F8-A892-6257EC470B6B wds:Q28609673-C8E8401F-6DF0-4E94-9091-6A83D09A358B wds:Q28609673-CA56D65C-21B2-4A6E-BFFD-26878ACCAEFB wds:Q28609673-CE6E37C4-E227-4CF3-A5A9-07AE103354BD wds:Q28609673-DF5B9C2D-40A3-4BE7-BEAE-7679031EBC5D wds:Q28609673-E161ED34-993A-4A48-BACA-ABBCB5050BAE wds:Q28609673-E5272A19-020E-4106-9A24-E2D64C35DDD3 wds:Q28609673-EBF5E48E-5991-4624-9B7A-F278816DF381 wds:Q28609673-EC68FC4A-DD45-4338-BDCB-8F4AB71FBF61 wds:Q28609673-EE8C1A85-F4B2-470A-9D83-9137FD6E9BA9 wds:Q28609673-F08EFB12-6B64-4D66-B941-8BA3107FD146 wds:Q28609673-F172F000-1BD2-4292-BEB4-AF8EFEF78B80 wds:Q28609673-F1829208-5D2F-4862-BB40-D6FFC6D1DD14 wds:Q28609673-F7362FE5-966E-4BAA-9831-E7F4FB6CC993 wds:Q28609673-FD83C8EF-27C7-4304-833E-358C8C98ACD8 wds:Q28609673-FD85EB83-803A-42AA-BB25-A27BEFEA3AC4 wds:Q28609673-FDE04BED-27CE-4015-8E77-20BD09757CD5
cites work	Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia Mutations in the human Jagged1 gene are responsible for Alagille syndrome Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1 Delta-1 activation of notch-1 signaling results in HES-1 transactivation. Two transmembrane aspartates in presenilin-1 required for presenilin endoproteolysis and gamma-secretase activity Neoplastic transformation by truncated alleles of human NOTCH1/TAN1 and NOTCH2. Calcium binding to tandem repeats of EGF-like modules. Expression and characterization of the EGF-like modules of human Notch-1 implicated in receptor-ligand interactions Molecular interactions between the protein products of the neurogenic loci Notch and Delta, two EGF-homologous genes in Drosophila Signal transduction by activated mNotch: importance of proteolytic processing and its regulation by the extracellular domain. Expression of an extracellular deletion of Xotch diverts cell fate in Xenopus embryos. The Notch1 receptor is cleaved constitutively by a furin-like convertase The notch 3 intracellular domain represses notch 1-mediated activation through Hairy/Enhancer of split (HES) promoters Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 Isolation and functional analysis of a cDNA for human Jagged2, a gene encoding a ligand for the Notch1 receptor TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms Notch-1 signalling requires ligand-induced proteolytic release of intracellular domain Signalling downstream of activated mammalian Notch Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain Nuclear access and action of notch in vivo Specific EGF repeats of Notch mediate interactions with Delta and serrate: Implications for notch as a multifunctional receptor Requirement for dynamin during Notch signaling in Drosophila neurogenesis. Analysis of gain-of-function mutations of the lin-12 gene of Caenorhabditis elegans. Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch alleles Oncogenic forms of NOTCH1 lacking either the primary binding site for RBP-Jkappa or nuclear localization sequences retain the ability to associate with RBP-Jkappa and activate transcription Notch inhibition of E47 supports the existence of a novel signaling pathway Correlating notch signaling with thymocyte maturation Notch signaling: cell fate control and signal integration in development Intracellular cleavage of Notch leads to a heterodimeric receptor on the plasma membrane Antineurogenic phenotypes induced by truncated Notch proteins indicate a role in signal transduction and may point to a novel function for Notch in nuclei An activated Notch receptor blocks cell-fate commitment in the developing Drosophila eye. Serrate expression can functionally replace Delta activity during neuroblast segregation in the Drosophila embryo. Indirect evidence for Delta-dependent intracellular processing of notch in Drosophila embryos The folding and structural integrity of the first LIN-12 module of human Notch1 are calcium-dependent. Complex proteolytic processing acts on Delta, a transmembrane ligand for Notch, during Drosophila development

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