| Attributes | Values |
|---|
| rdf:type
| |
| description
| - artículu científicu espublizáu en 2003 (ast)
- vědecký článek publikovaný v roce 2003 (cs)
- 2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած (hyw)
- مقالة علمية (نشرت في ديسمبر 2003) (ar)
- наукова стаття, опублікована в грудні 2003 (uk)
- مقالة علمية تنشرت ف 1 دجنبر 2003 (ary)
- 2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված (hy)
- wetenschappelijk artikel (gepubliceerd op 2003/12/01) (nl)
- vedecký článok (publikovaný 2003/12/01) (sk)
- article scientifique publié en 2003 (fr)
- im Dezember 2003 veröffentlichter wissenschaftlicher Artikel (de)
- scientific journal article (en)
|
| publication date
| |
| publication date
| |
| language of work or name
| |
| language of work or name
| |
| author name string
| |
| OpenCitations bibliographic resource ID
| |
| OpenCitations bibliographic resource ID
| |
| author name string
| - M. Beth Goens
- Mary K. Walker
|
| OpenCitations bibliographic resource ID
| |
| rdfs:label
| - Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (en)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (nl)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (ast)
|
| skos:prefLabel
| - Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (en)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (nl)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (ast)
|
| name
| - Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (en)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (nl)
- Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (ast)
|
| author
| |
| author
| |
| title
| |
| title
| - Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure (en)
|
| page(s)
| |
| page(s)
| |
| instance of
| |
| instance of
| |
| main subject
| |
| main subject
| |
| PubMed ID
| |
| PubMed ID
| |
| PubMed ID
| |
| published in
| |
| published in
| |
| issue
| |
| volume
| |
| issue
| |
| volume
| |
| DOI
| |
| DOI
| |
| DOI
| - 10.1016/J.TAAP.2003.08.008
|
| Fatcat ID
| |
| Fatcat ID
| - release_k4hajqyw2vck7j2rvqlm37nsv4
|
| is about
of | |
| is cites work
of | - The aryl hydrocarbon receptor: a perspective on potential roles in the immune system
- Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infection
- Effects of trans-resveratrol on hypertension-induced cardiac hypertrophy using the partially nephrectomized rat model.
- Ah Receptor Pathway Intricacies; Signaling Through Diverse Protein Partners and DNA-Motifs
- The role of aryl hydrocarbon receptor in the pathogenesis of cardiovascular diseases.
- CYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substrates
- Dioxin receptor deficiency impairs angiogenesis by a mechanism involving VEGF-A depletion in the endothelium and transforming growth factor-beta overexpression in the stroma.
- Nonadditive effects of PAHs on Early Vertebrate Development: mechanisms and implications for risk assessment
- Aryl hydrocarbon receptor is activated by glucose and regulates the thrombospondin-1 gene promoter in endothelial cells
- AhR expression and polymorphisms are associated with risk of coronary arterial disease in Chinese population
- The role of endogenous aryl hydrocarbon receptor signaling in cardiovascular physiology
- New Trends in Aryl Hydrocarbon Receptor Biology
- Role of the aryl hydrocarbon receptor in carcinogenesis and potential as a drug target
- Inhibition of cytochrome P4501-dependent clearance of the endogenous agonist FICZ as a mechanism for activation of the aryl hydrocarbon receptor.
- Diet-relevant phytochemical intake affects the cardiac AhR and nrf2 transcriptome and reduces heart failure in hypertensive rats
- Endothelin-1-mediated increase in reactive oxygen species and NADPH Oxidase activity in hearts of aryl hydrocarbon receptor (AhR) null mice.
- Dioxin exposure disrupts the differentiation of mouse embryonic stem cells into cardiomyocytes
- Identification and characterization of genes susceptible to transcriptional cross-talk between the hypoxia and dioxin signaling cascades
- An activated renin-angiotensin system maintains normal blood pressure in aryl hydrocarbon receptor heterozygous mice but not in null mice
- Cytochrome P4501A1 is required for vascular dysfunction and hypertension induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin
- Endothelial cell-specific aryl hydrocarbon receptor knockout mice exhibit hypotension mediated, in part, by an attenuated angiotensin II responsiveness
- Hypoxia-inducible factor 2α (HIF-2α) heterozygous-null mice exhibit exaggerated carotid body sensitivity to hypoxia, breathing instability, and hypertension
- Aryl hydrocarbon receptor deletion in cerebellar granule neuron precursors impairs neurogenesis
- Molecular mechanisms of 2,3,7,8-tetrachlorodibenzo-p-dioxin cardiovascular embryotoxicity
- Using U0126 to dissect the role of the extracellular signal-regulated kinase 1/2 (ERK1/2) cascade in the regulation of gene expression by endothelin-1 in cardiac myocytes.
- Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries
- Captopril normalizes insulin signaling and insulin-regulated substrate metabolism in obese (ob/ob) mouse hearts.
- Perspectives on the potential involvement of the AH receptor-dioxin axis in cardiovascular disease
- Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte Functions
- Disruption of aryl hydrocarbon receptor homeostatic levels during embryonic stem cell differentiation alters expression of homeobox transcription factors that control cardiomyogenesis
- Transcriptional factor aryl hydrocarbon receptor (Ahr) controls cardiovascular and respiratory functions by regulating the expression of the Vav3 proto-oncogene
- Crosstalk between the aryl hydrocarbon receptor and hypoxia on the constitutive expression of cytochrome P4501A1 mRNA.
- The mouse and human Ah receptor differ in recognition of LXXLL motifs
- Polychlorinated biphenyl 77 augments angiotensin II-induced atherosclerosis and abdominal aortic aneurysms in male apolipoprotein E deficient mice.
- NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice
- Captopril therapy decreases both expression and function of alpha-adrenoceptors in pre- hypertensive rat aorta
- Baicalin reduces blood lipids and inflammation in patients with coronary artery disease and rheumatoid arthritis: a randomized, double-blind, placebo-controlled trial.
- Pluripotency factors and Polycomb Group proteins repress aryl hydrocarbon receptor expression in murine embryonic stem cells
|
![[RDF Data]](/fct/images/sw-rdf-blue.png)
![[cxml]](/fct/images/cxml_doc.png)
![[csv]](/fct/images/csv_doc.png)
![[text]](/fct/images/ntriples_doc.png)
![[turtle]](/fct/images/n3turtle_doc.png)
![[ld+json]](/fct/images/jsonld_doc.png)
![[rdf+json]](/fct/images/json_doc.png)
![[rdf+xml]](/fct/images/xml_doc.png)
![[atom+xml]](/fct/images/atom_doc.png)
![[html]](/fct/images/html_doc.png)