About: William R Heath

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) araştırmacı (tr) wetenschapper (nl) дослідник (uk) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	William R Heath (en) William R Heath (es) William R Heath (nl) William R Heath (sl) William R Heath (ast)
skos:prefLabel	William R Heath (en) William R Heath (es) William R Heath (nl) William R Heath (sl) William R Heath (ast)
name	William R Heath (en) William R Heath (es) William R Heath (nl) William R Heath (sl) William R Heath (ast)
given name	wds:Q41610442-6DF88506-B42F-4782-91BC-8FFAD8F0654C
given name	William
instance of	wds:Q41610442-BBFF9445-302A-4388-9750-B3EA04F9A0E6
instance of	Homo sapiens
award received	wds:Q41610442-b02ef491-4c1a-83b1-a704-b07eb413f5db
award received	Fellow of the Australian Academy of Science
Google Scholar author ID	wds:Q41610442-a2d2f3ea-48af-49a3-2c99-5c7ef4253663
Google Scholar author ID	WdUHeYYAAAAJ
skos:altLabel	William Heath (en) Bill Heath (en)
occupation	wds:Q41610442-4CF966A6-B42F-4157-95B6-55D72D6439C0
employer	wds:Q41610442-B1FDA147-CC42-49E9-A5AB-5706E43BBEDB
sex or gender	wds:Q41610442-9834AB82-0AC5-40A5-8864-EFC346FF51CC
family name	wds:Q41610442-555638E4-20C4-4F61-8A9A-465AE5381647
occupation	researcher
employer	University of Melbourne
sex or gender	male
family name	Heath
ORCID iD	wds:Q41610442-A8D4BB80-B2B1-4141-8105-146318403863
ORCID iD	https://orcid.org/0000-0001-9670-259X
ORCID iD	0000-0001-9670-259X
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q41610442
is author of	Most lymphoid organ dendritic cell types are phenotypically and functionally immature. Induction of T-cell-mediated skin disease specific for antigen transgenically expressed in keratinocytes Immunity or tolerance? That is the question for dendritic cells Different patterns of peripheral migration by memory CD4+ and CD8+ T cells. Multiple dendritic cell populations activate CD4+ T cells after viral stimulation Defective TCR expression in transgenic mice constructed using cDNA-based α- and β-chain genes under the control of heterologous regulatory elements The skin-resident and migratory immune system in steady state and memory: innate lymphocytes, dendritic cells and T cells Spatiotemporally Distinct Interactions with Dendritic Cell Subsets Facilitates CD4+ and CD8+ T Cell Activation to Localized Viral Infection Progression of armed CTL from draining lymph node to spleen shortly after localized infection with herpes simplex virus 1. Normal proportion and expression of maturation markers in migratory dendritic cells in the absence of germs or Toll-like receptor signaling Dangerous liaisons CD8+ T cells from a novel T cell receptor transgenic mouse induce liver-stage immunity that can be boosted by blood-stage infection in rodent malaria Cross-presentation in viral immunity and self-tolerance. Latent infection with herpes simplex virus is associated with ongoing CD8+ T-cell stimulation by parenchymal cells within sensory ganglia A new IFN-like cytokine, limitin, modulates the immune response without influencing thymocyte development Help for cytotoxic-T-cell responses is mediated by CD40 signalling. Distinct migrating and nonmigrating dendritic cell populations are involved in MHC class I-restricted antigen presentation after lung infection with virus Epidermal viral immunity induced by CD8alpha+ dendritic cells but not by Langerhans cells The dominant role of CD8+ dendritic cells in cross-presentation is not dictated by antigen capture Systemic activation of dendritic cells by Toll-like receptor ligands or malaria infection impairs cross-presentation and antiviral immunity Cutting edge: conventional CD8 alpha+ dendritic cells are generally involved in priming CTL immunity to viruses. Migratory Dendritic Cells Transfer Antigen to a Lymph Node-Resident Dendritic Cell Population for Efficient CTL Priming Antigen-specific CD8+ T cell subset distribution in lymph nodes draining the site of herpes simplex virus infection A specific anti-Aire antibody reveals aire expression is restricted to medullary thymic epithelial cells and not expressed in periphery. Induction of tumor cell apoptosis in vivo increases tumor antigen cross-presentation, cross-priming rather than cross-tolerizing host tumor-specific CD8 T cells Differential MHC class II synthesis and ubiquitination confers distinct antigen-presenting properties on conventional and plasmacytoid dendritic cells Peripheral deletion of autoreactive CD8+ T cells in transgenic mice expressing H-2Kb in the liver The molecular signature of CD8+ T cells undergoing deletional tolerance Expression of two T cell receptor α chains on the surface of normal murine T cells Blood-stage Plasmodium infection induces CD8+ T lymphocytes to parasite-expressed antigens, largely regulated by CD8alpha+ dendritic cells Aire-deficient C57BL/6 mice mimicking the common human 13-base pair deletion mutation present with only a mild autoimmune phenotype Peptide antagonists that promote positive selection are inefficient at T cell activation and thymocyte deletion SOCS1: a potent and multifaceted regulator of cytokines and cell-mediated inflammation. Cross-priming: its beginnings. Equivalent stimulation of naive and memory CD8 T cells by DNA vaccination: a dendritic cell-dependent process. Dendritic cell preactivation impairs MHC class II presentation of vaccines and endogenous viral antigens Life cycle, migration and antigen presenting functions of spleen and lymph node dendritic cells: limitations of the Langerhans cells paradigm Cross-presentation, dendritic cell subsets, and the generation of immunity to cellular antigens Cross-presentation of viral and self antigens by skin-derived CD103+ dendritic cells Memory T cells in nonlymphoid tissue that provide enhanced local immunity during infection with herpes simplex virus DEC-205 is a cell surface receptor for CpG oligonucleotides The dendritic cell subtype-restricted C-type lectin Clec9A is a target for vaccine enhancement Memory T cell subsets, migration patterns, and tissue residence The developmental pathway for CD103(+)CD8+ tissue-resident memory T cells of skin. Cutting edge: central memory T cells do not show accelerated proliferation or tissue infiltration in response to localized herpes simplex virus-1 infection Characterization of the ovalbumin-specific TCR transgenic line OT-I: MHC elements for positive and negative selection Protective immunity to liver-stage malaria Persistence of skin-resident memory T cells within an epidermal niche

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